David S. Park, Ph.D.

Affiliations: 
University of Ottawa, Ottawa, ON, Canada 
Area:
Cell death
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"David Park"
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Publications

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Huang T, González YR, Qu D, et al. (2019) The pro-death role of Cited2 in stroke is regulated by E2F1/4 transcription factors. The Journal of Biological Chemistry
Yang J, Kim KS, Iyirhiaro GO, et al. (2019) DJ-1 modulates the unfolded protein response and cell death via upregulation of ATF4 following ER stress. Cell Death & Disease. 10: 135
Boonying W, Joselin A, Huang E, et al. (2019) Pink1 regulates FKBP5 interaction with AKT/PHLPP and protects neurons from neurotoxin stress induced by MPP. Journal of Neurochemistry
Kim KS, Marcogliese PC, Yang J, et al. (2018) Regulation of myeloid cell phagocytosis by LRRK2 via WAVE2 complex stabilization is altered in Parkinson's disease. Proceedings of the National Academy of Sciences of the United States of America
Giguere N, Pacelli C, Saumure C, et al. (2018) Comparative analysis of Parkinson's disease-associated genes reveals altered survival and bioenergetics of parkin-deficient dopamine neurons in mice. The Journal of Biological Chemistry
Connolly NMC, Theurey P, Adam-Vizi V, et al. (2017) Guidelines on experimental methods to assess mitochondrial dysfunction in cellular models of neurodegenerative diseases. Cell Death and Differentiation
Connolly NMC, Theurey P, Adam-Vizi V, et al. (2017) Guidelines on experimental methods to assess mitochondrial dysfunction in cellular models of neurodegenerative diseases. Cell Death and Differentiation
Huang E, Qu D, Huang T, et al. (2017) PINK1-mediated phosphorylation of LETM1 regulates mitochondrial calcium transport and protects neurons against mitochondrial stress. Nature Communications. 8: 1399
Iyirhiaro GO, Soon Im D, Boonying W, et al. (2017) Cdc25A is a critical mediator of ischemic neuronal death in vitro and in vivo. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Khacho M, Clark A, Svoboda DS, et al. (2017) Mitochondrial dysfunction underlies cognitive defects as a result of neural stem cell depletion and impaired neurogenesis. Human Molecular Genetics
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