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Qing Yang

Affiliations: 
Integrative Biology and Pharmacology University of Texas-Houson, Houston, TX, United States 
Area:
pain mechanisms
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"Qing Yang"
Mean distance: 14.48 (cluster 6)
 		SNBCP
Cross-listing: Chemistry Tree

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Edgar T. Walters research scientist 2007-2011 University of Texas Health Science Center Houston

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Publications

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Wu Z, Toro G, Xu G, et al. (2022) Paclitaxel Inhibits KCNQ Channels in Primary Sensory Neurons to Initiate the Development of Painful Peripheral Neuropathy. Cells. 11
Wu Z, Li L, Xie F, et al. (2020) Enhancing KCNQ channel activity improves neurobehavioral recovery after spinal cord injury. The Journal of Pharmacology and Experimental Therapeutics
Li L, Li J, Zuo Y, et al. (2018) Activation of KCNQ channels prevents paclitaxel-induced peripheral neuropathy and associated neuropathic pain. The Journal of Pain : Official Journal of the American Pain Society
Zhang H, Li Y, Yang Q, et al. (2018) Morphological and Physiological Plasticity of Spinal Lamina II GABA Neurons Is Induced by Sciatic Nerve Chronic Constriction Injury in Mice. Frontiers in Cellular Neuroscience. 12: 143
Wu Z, Li L, Xie F, et al. (2017) Activation of KCNQ channels suppresses spontaneous activity in DRG neurons and reduces chronic pain after spinal cord injury. Journal of Neurotrauma
Bavencoffe A, Li Y, Wu Z, et al. (2016) Persistent Electrical Activity in Primary Nociceptors after Spinal Cord Injury Is Maintained by Scaffolded Adenylyl Cyclase and Protein Kinase A and Is Associated with Altered Adenylyl Cyclase Regulation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 1660-8
Yang Q, Wu Z, Hadden JK, et al. (2014) Persistent pain after spinal cord injury is maintained by primary afferent activity. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 10765-9
Yin S, Luo J, Qian A, et al. (2013) Retinoids activate the irritant receptor TRPV1 and produce sensory hypersensitivity. The Journal of Clinical Investigation. 123: 3941-51
Wu Z, Yang Q, Crook RJ, et al. (2013) TRPV1 channels make major contributions to behavioral hypersensitivity and spontaneous activity in nociceptors after spinal cord injury. Pain. 154: 2130-41
Bedi SS, Yang Q, Crook RJ, et al. (2010) Chronic spontaneous activity generated in the somata of primary nociceptors is associated with pain-related behavior after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 14870-82
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