Brian S. Tanaka, Post-doc
Affiliations: | 2006-2014 | Microbiology & Molecular Genetics | University of California, Irvine, Irvine, CA |
2014- | Neurology | Yale University, New Haven, CT |
Area:
epilepsy, sodium channel, painGoogle:
"Brian Tanaka"Mean distance: 14.05 (cluster 11) | S | N | B | C | P |
Cross-listing: Chemistry Tree
Parents
Sign in to add mentorAlan L. Goldin | grad student | 2006-2014 | UC Irvine | |
(Reduced Scn8a Expression in Excitatory Neurons Decreases Seizure Susceptibility.) | ||||
Stephen G. Waxman | post-doc | 2014- | Yale |
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Publications
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Yuan JH, Estacion M, Mis MA, et al. (2021) variants and pain modulation: a missense variant in Kv7.3 contributes to pain resilience. Brain Communications. 3: fcab212 |
McMahon KL, Tay B, Deuis JR, et al. (2020) Pharmacological activity and NMR solution structure of the leech peptide HSTX-I. Biochemical Pharmacology. 114082 |
Labau JI, Estacion M, Tanaka BS, et al. (2020) Differential effect of lacosamide on Nav1.7 variants from responsive and non-responsive patients with small fibre neuropathy. Brain : a Journal of Neurology |
Urru M, Muzzi M, Coppi E, et al. (2019) Dexpramipexole blocks Nav1.8 sodium channels and provides analgesia in multiple nociceptive and neuropathic pain models. Pain |
Akin EJ, Higerd GP, Mis MA, et al. (2019) Building sensory axons: Delivery and distribution of Na1.7 channels and effects of inflammatory mediators. Science Advances. 5: eaax4755 |
Israel MR, Tanaka BS, Castro J, et al. (2019) Na 1.6 regulates excitability of mechanosensitive sensory neurons. The Journal of Physiology |
Vrselja Z, Daniele SG, Silbereis J, et al. (2019) Restoration of brain circulation and cellular functions hours post-mortem. Nature. 568: 336-343 |
Mis MA, Yang Y, Tanaka BS, et al. (2018) Resilience to Pain: A Peripheral Component Identified using induced Pluripotent Stem Cells and Dynamic Clamp. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Huang J, Mis MA, Tanaka B, et al. (2018) Atypical changes in DRG neuron excitability and complex pain phenotype associated with a Nav1.7 mutation that massively hyperpolarizes activation. Scientific Reports. 8: 1811 |
Tanaka BS, Nguyen PT, Zhou EY, et al. (2017) Gain-of-Function Mutation of a Voltage-Gated Sodium Channel NaV1.7 Associated with Peripheral Pain and Impaired Limb Development. The Journal of Biological Chemistry |