Gustavo A. Rodriguez, Ph.D.
Affiliations: | 2015- | Neuroscience | Columbia University, New York, NY |
Area:
neuronal network dysfunction in ADGoogle:
"Gustavo Rodriguez"Mean distance: 17.08 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentorG. William Rebeck | grad student | 2009- | Georgetown |
Karen Duff | post-doc | 2015- | Columbia University Medical School |
Abid Hussaini | post-doc | 2015- | Columbia University Medical School |
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Publications
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Rodriguez GA, Barrett GM, Duff KE, et al. (2020) Chemogenetic attenuation of neuronal activity in the entorhinal cortex reduces Aβ and tau pathology in the hippocampus. Plos Biology. 18: e3000851 |
Fu H, Rodriguez GA, Herman M, et al. (2017) Tau Pathology Induces Excitatory Neuron Loss, Grid Cell Dysfunction, and Spatial Memory Deficits Reminiscent of Early Alzheimer's Disease. Neuron |
Wu JW, Hussaini SA, Bastille IM, et al. (2016) Neuronal activity enhances tau propagation and tau pathology in vivo. Nature Neuroscience |
Rodriguez GA, Tai LM, LaDu MJ, et al. (2014) Human APOE4 increases microglia reactivity at Aβ plaques in a mouse model of Aβ deposition. Journal of Neuroinflammation. 11: 111 |
Zhao W, Dumanis SB, Tamboli IY, et al. (2014) Human APOE genotype affects intraneuronal Aβ1-42 accumulation in a lentiviral gene transfer model. Human Molecular Genetics. 23: 1365-75 |
Rodriguez GA, Burns MP, Weeber EJ, et al. (2013) Young APOE4 targeted replacement mice exhibit poor spatial learning and memory, with reduced dendritic spine density in the medial entorhinal cortex. Learning & Memory (Cold Spring Harbor, N.Y.). 20: 256-66 |
Rodriguez GA, Hussaini SA, Duff KE. (2006) P1-022: ATTENUATION OF Aβ-ASSOCIATED HYPERACTIVITY IN THE ENTORHINAL CORTEX AMELIORATES HIPPOCAMPAL PLAQUE BURDEN Alzheimer's & Dementia. 14: P273-P273 |