Yedy Israel
Affiliations: | University of Chile, Santiago, Región Metropolitana, Chile |
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Publications
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| Quintanilla ME, Israel Y. (2023) Role of Metabolism on Alcohol Preference, Addiction, and Treatment. Current Topics in Behavioral Neurosciences |
| Ezquer F, Quintanilla ME, Morales P, et al. (2022) A dual treatment blocks alcohol binge-drinking relapse: Microbiota as a new player. Drug and Alcohol Dependence. 236: 109466 |
| Quintanilla ME, Ezquer F, Morales P, et al. (2022) A dual mechanism fully blocks ethanol relapse: Role of vagal innervation. Addiction Biology. 27: e13140 |
| Ezquer F, Quintanilla ME, Moya-Flores F, et al. (2021) Innate gut microbiota predisposes to high alcohol consumption. Addiction Biology. e13018 |
| Quintanilla ME, Ezquer F, Morales P, et al. (2020) N-Acetylcysteine and Acetylsalicylic Acid Inhibit Alcohol Consumption by Different Mechanisms: Combined Protection. Frontiers in Behavioral Neuroscience. 14: 122 |
| Israel Y, Quintanilla ME, Ezquer F, et al. (2019) Aspirin and N-acetylcysteine co-administration markedly inhibit chronic ethanol intake and block relapse binge drinking: Role of neuroinflammation-oxidative stress self-perpetuation. Addiction Biology. e12853 |
| Quintanilla ME, Ezquer F, Morales P, et al. (2019) Intranasal mesenchymal stem cell secretome administration markedly inhibits alcohol and nicotine self-administration and blocks relapse-intake: mechanism and translational options. Stem Cell Research & Therapy. 10: 205 |
| Rivera-Meza M, Vásquez D, Quintanilla ME, et al. (2018) Activation of mitochondrial aldehyde dehydrogenase (ALDH2) by ALDA-1 reduces both the acquisition and maintenance of ethanol intake in rats: A dual mechanism? Neuropharmacology |
| Ezquer F, Quintanilla ME, Morales P, et al. (2018) Intranasal delivery of mesenchymal stem cell-derived exosomes reduces oxidative stress and markedly inhibits ethanol consumption and post-deprivation relapse drinking. Addiction Biology |
| Quintanilla ME, Morales P, Ezquer F, et al. (2018) Commonality of ethanol and nicotine reinforcement and relapse in Wistar-derived UChB rats: inhibition by N-acetyl cysteine. Alcoholism, Clinical and Experimental Research |