Thomas M. Maynard

Affiliations: 
George Washington University, Washington, DC, United States 
Area:
22q11 Deletion Syndrome, cortical development, neural crest
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"Thomas Maynard"
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Motahari Z, Maynard TM, Popratiloff A, et al. (2020) Aberrant Early Growth of Individual Trigeminal Sensory and Motor Axons in a Series of Mouse Genetic Models of 22q11.2 Deletion Syndrome. Human Molecular Genetics
Yitsege G, Stokes BA, Sabatino JA, et al. (2020) Variations in maternal vitamin A intake modifies phenotypes in a mouse model of 22q11.2 deletion syndrome. Birth Defects Research
Maynard TM, Horvath A, Bernot JP, et al. (2020) Transcriptional dysregulation in developing trigeminal sensory neurons in the LgDel mouse model of DiGeorge 22q11.2 deletion syndrome. Human Molecular Genetics
Maynard TM, Zohn IE, Moody SA, et al. (2020) Suckling, Feeding, and Swallowing: Behaviors, Circuits, and Targets for Neurodevelopmental Pathology. Annual Review of Neuroscience
Welby L, Caudill H, Yitsege G, et al. (2020) Persistent Feeding and Swallowing Deficits in a Mouse Model of 22q11.2 Deletion Syndrome. Frontiers in Neurology. 11: 4
Maynard TM, Horvath A, Bernout J, et al. (2020) Transcriptional dysregulation in developing trigeminal sensory neurons in the LgDel mouse model of DiGeorge 22q11.2 Deletion Syndrome. Human Molecular Genetics
Motahari Z, Moody SA, Maynard TM, et al. (2019) In the line-up: deleted genes associated with DiGeorge/22q11.2 deletion syndrome: are they all suspects? Journal of Neurodevelopmental Disorders. 11: 7
Fernandez A, Meechan DW, Karpinski BA, et al. (2019) Mitochondrial Dysfunction Leads to Cortical Under-Connectivity and Cognitive Impairment. Neuron
Steullet P, Cabungcal JH, Coyle J, et al. (2017) Oxidative stress-driven parvalbumin interneuron impairment as a common mechanism in models of schizophrenia. Molecular Psychiatry
Sherman JH, Karpinski BA, Fralish MS, et al. (2017) Foxd4 is essential for establishing neural cell fate and for neuronal differentiation. Genesis (New York, N.Y. : 2000)
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