Thomas M. Maslanik, Ph.D.
Affiliations: | 2012 | Integrative Physiology | University of Colorado, Boulder, Boulder, CO, United States |
Area:
Physiology Biology, ImmunologyGoogle:
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Parents
Sign in to add mentor| Monika R. Fleshner | grad student | 2012 | CU Boulder | |
| (Defining stress-induced systemic sterile inflammatory responses: Network, signals, and pathways.) | ||||
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| Beninson LA, Brown PN, Loughridge AB, et al. (2014) Acute stressor exposure modifies plasma exosome-associated heat shock protein 72 (Hsp72) and microRNA (miR-142-5p and miR-203). Plos One. 9: e108748 |
| Thompson RS, Strong PV, Clark PJ, et al. (2014) Repeated fear-induced diurnal rhythm disruptions predict PTSD-like sensitized physiological acute stress responses in F344 rats. Acta Physiologica (Oxford, England). 211: 447-65 |
| Speaker KJ, Cox SS, Paton MM, et al. (2014) Six weeks of voluntary wheel running modulates inflammatory protein (MCP-1, IL-6, and IL-10) and DAMP (Hsp72) responses to acute stress in white adipose tissue of lean rats. Brain, Behavior, and Immunity. 39: 87-98 |
| Thompson RS, Christianson JP, Maslanik TM, et al. (2013) Effects of stressor controllability on diurnal physiological rhythms. Physiology & Behavior. 112: 32-9 |
| Maslanik T, Mahaffey L, Tannura K, et al. (2013) The inflammasome and danger associated molecular patterns (DAMPs) are implicated in cytokine and chemokine responses following stressor exposure Brain, Behavior, and Immunity. 28: 54-62 |
| Maslanik T, Tannura K, Mahaffey L, et al. (2013) Correction: Commensal Bacteria and MAMPs Are Necessary for Stress-Induced Increases in IL-1β and IL-18 but Not IL-6, IL-10 or MCP-1 Plos One. 8 |
| Maslanik T, Tannura K, Mahaffey L, et al. (2012) Commensal bacteria and MAMPs are necessary for stress-induced increases in IL-1β and IL-18 but not IL-6, IL-10 or MCP-1. Plos One. 7: e50636 |
| Hutchinson MR, Northcutt AL, Hiranita T, et al. (2012) Opioid activation of toll-like receptor 4 contributes to drug reinforcement. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 11187-200 |
| Campisi J, Sharkey C, Johnson JD, et al. (2012) Stress-induced facilitation of host response to bacterial challenge in F344 rats is dependent on extracellular heat shock protein 72 and independent of alpha beta T cells. Stress (Amsterdam, Netherlands). 15: 637-46 |
| Maslanik T, Bernstein-Hanley I, Helwig B, et al. (2012) The impact of acute-stressor exposure on splenic innate immunity: A gene expression analysis Brain, Behavior, and Immunity. 26: 142-149 |