John E. Chad

Affiliations: 
University of Southampton (UK), Southampton, England, United Kingdom 
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Bailey JL, O'Connor V, Hannah M, et al. (2011) In vitro CNS tissue analogues formed by self-organisation of reaggregated post-natal brain tissue. Journal of Neurochemistry. 117: 1020-32
Pilkington B, Cuttle M, Chad J, et al. (2001) Localization of presynaptic metabotropic glutamate receptors at the Calyces of Held Biochemical Society Transactions. 29
Chen Y, Chad JE, Cannon RC, et al. (1999) Reduced Mg2+ blockade of synaptically activated N-methyl-D-aspartate receptor-channels in CA1 pyramidal neurons in kainic acid-lesioned rat hippocampus. Neuroscience. 88: 727-39
Wheal HV, Chad JE, Cannon RC, et al. (1998) Pro-epileptic changes in synaptic function can be accompanied by pro- epileptic changes in neuronal excitability Trends in Neurosciences. 21: 167-174
Chen Y, Chad JE, Wheal HV. (1997) Synaptic release rather than failure in the conditioning pulse results in paired-pulse facilitation during minimal synaptic stimulation in the rat hippocampal CA1 neurones. Neuroscience Letters. 218: 204-8
Caddick SJ, Stanford IM, Chad JE. (1995) 2-Hydroxy-saclofen causes a phaclofen-reversible reduction in population spike amplitude in the rat hippocampal slice. European Journal of Pharmacology. 274: 41-6
Stanford IM, Wheal HV, Chad JE. (1995) Bicuculline enhances the late GABAB receptor-mediated paired-pulse inhibition observed in rat hippocampal slices. European Journal of Pharmacology. 277: 229-34
Chad JE, Eckert R. (1986) An enzymatic mechanism for calcium current inactivation in dialysed Helix neurones The Journal of Physiology. 378: 31-51
Chad JE, Eckert R. (1984) Calcium domains associated with individual channels can account for anomalous voltage relations of CA-dependent responses Biophysical Journal. 45: 993-999
Chad J, Eckert R, Ewald D. (1984) Kinetics of calcium‐dependent inactivation of calcium current in voltage‐clamped neurones of Aplysia californica The Journal of Physiology. 347: 279-300
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