Rakez Kayed
Affiliations: | Neurology | University of Texas Medical Branch, Galveston, TX |
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Publications
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Shi H, Mirzaei N, Koronyo Y, et al. (2024) Identification of retinal tau oligomers, citrullinated tau, and other tau isoforms in early and advanced AD and relations to disease status. Biorxiv : the Preprint Server For Biology |
Bhatt N, Puangmalai N, Sengupta U, et al. (2024) C9orf72-associated dipeptide protein repeats form A11-positive oligomers in amyotrophic lateral sclerosis and frontotemporal dementia. The Journal of Biological Chemistry. 300: 105628 |
Puangmalai N, Bhatt N, Bittar A, et al. (2023) Traumatic brain injury derived pathological tau polymorphs induce the distinct propagation pattern and neuroinflammatory response in wild type mice. Progress in Neurobiology. 232: 102562 |
Gaikwad S, Senapati S, Haque MA, et al. (2023) Senescence, brain inflammation, and oligomeric tau drive cognitive decline in Alzheimer's disease: Evidence from clinical and preclinical studies. Alzheimer's & Dementia : the Journal of the Alzheimer's Association |
Solorzano A, Brady M, Bhatt N, et al. (2023) Central and peripheral tau retention modulated by an anti-tau antibody. Biorxiv : the Preprint Server For Biology |
Bhopatkar AA, Kayed R. (2023) Flanking regions, amyloid cores, and polymorphism: the potential interplay underlying structural diversity. The Journal of Biological Chemistry. 299: 105122 |
Arar S, Haque MA, Kayed R. (2023) Protein aggregation and neurodegenerative disease: Structural outlook for the novel therapeutics. Proteins |
Hussong SA, Banh AQ, Van Skike CE, et al. (2023) Soluble pathogenic tau enters brain vascular endothelial cells and drives cellular senescence and brain microvascular dysfunction in a mouse model of tauopathy. Nature Communications. 14: 2367 |
Luciani M, Montalbano M, Troncone L, et al. (2023) Big tau aggregation disrupts microtubule tyrosination and causes myocardial diastolic dysfunction: from discovery to therapy. European Heart Journal. 44: 1560-1570 |
Dhakal S, Robang AS, Bhatt N, et al. (2022) Distinct neurotoxic TDP-43 fibril polymorphs are generated by heterotypic interactions with α-Synuclein. The Journal of Biological Chemistry. 102498 |