Nazim Kourdougli
Affiliations: | David Geffen School of Medicine at UCLA, Los Angeles, CA, United States |
Area:
development, fragile x syndrome; calcium imaging, somatosensory cortexGoogle:
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Publications
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| Kourdougli N, Nomura T, Wu M, et al. (2024) The NKCC1 inhibitor bumetanide restores cortical feedforward inhibition and lessens sensory hypersensitivity in early postnatal fragile X mice. Biological Psychiatry |
| Wu MW, Kourdougli N, Portera-Cailliau C. (2024) Network state transitions during cortical development. Nature Reviews. Neuroscience |
| Goel A, Cantu DA, Guilfoyle J, et al. (2023) Author Correction: Impaired perceptual learning in a mouse model of Fragile X syndrome is mediated by parvalbumin neuron dysfunction and is reversible. Nature Neuroscience |
| Kourdougli N, Suresh A, Liu B, et al. (2023) Improvement of sensory deficits in fragile X mice by increasing cortical interneuron activity after the critical period. Neuron |
| Kourdougli N, Portera-Cailliau C. (2023) aGABRacadabra: A surprising new role for GABA receptors in cortical development. Neuron. 111: 146-149 |
| Cantu DA, Wang B, Gongwer MW, et al. (2020) EZcalcium: Open-Source Toolbox for Analysis of Calcium Imaging Data. Frontiers in Neural Circuits. 14: 25 |
| Goel A, Cantu DA, Guilfoyle J, et al. (2018) Author Correction: Impaired perceptual learning in a mouse model of Fragile X syndrome is mediated by parvalbumin neuron dysfunction and is reversible. Nature Neuroscience |
| Goel A, Cantu DA, Guilfoyle J, et al. (2018) Impaired perceptual learning in a mouse model of Fragile X syndrome is mediated by parvalbumin neuron dysfunction and is reversible. Nature Neuroscience. 21: 1404-1411 |