Sudheendra N R Rao, MBBS, PhD

Affiliations: 
2013-2018 The Miami Project to Cure Paralysis University of Miami, Coral Gables, FL 
 2018- Neurobiology National Centre for Biological Sciences (NCBS) 
Area:
Neurodegeneration, Epilepsy, Neurotrauma
Website:
https://scholar.google.com/citations?user=XYYF1xIAAAAJ&hl
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"Sudheendra Rao"
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Publications

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Chim H, Salgado CJ, Miller NL, et al. (2019) Analysis of Epineurial Lidocaine Injection for Nerve Transfers in a Rat Sciatic Nerve Model. The Journal of Hand Surgery
Knott EP, Assi M, Rao SN, et al. (2017) Phosphodiesterase Inhibitors as a Therapeutic Approach to Neuroprotection and Repair. International Journal of Molecular Sciences. 18
Rao SN, Pearse DD. (2016) Regulating Axonal Responses to Injury: The Intersection between Signaling Pathways Involved in Axon Myelination and The Inhibition of Axon Regeneration. Frontiers in Molecular Neuroscience. 9: 33
Rao S, Morales AA, Pearse DD. (2015) The Comparative Utility of Viromer RED and Lipofectamine for Transient Gene Introduction into Glial Cells. Biomed Research International. 2015: 458624
Sharma J, Mukherjee D, Rao SN, et al. (2013) Neuronatin-mediated aberrant calcium signaling and endoplasmic reticulum stress underlie neuropathology in Lafora disease. The Journal of Biological Chemistry. 288: 9482-90
Sharma J, Rao SN, Shankar SK, et al. (2011) Lafora disease ubiquitin ligase malin promotes proteasomal degradation of neuronatin and regulates glycogen synthesis. Neurobiology of Disease. 44: 133-41
Rao SN, Maity R, Sharma J, et al. (2010) Sequestration of chaperones and proteasome into Lafora bodies and proteasomal dysfunction induced by Lafora disease-associated mutations of malin. Human Molecular Genetics. 19: 4726-34
Rao SN, Sharma J, Maity R, et al. (2010) Co-chaperone CHIP stabilizes aggregate-prone malin, a ubiquitin ligase mutated in Lafora disease. The Journal of Biological Chemistry. 285: 1404-13
Godavarthi SK, Narender D, Mishra A, et al. (2009) Induction of chemokines, MCP-1, and KC in the mutant huntingtin expressing neuronal cells because of proteasomal dysfunction. Journal of Neurochemistry. 108: 787-95
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