XiaoYong Tong

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2002-2005 New York University School of Medicine, New York, NY, United States 
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"XiaoYong Tong"
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Cao X, Mao M, Diao J, et al. (2021) Ectopic Overexpression of PPARγ2 in the Heart Determines Differences in Hypertrophic Cardiomyopathy After Treatment With Different Thiazolidinediones in a Mouse Model of Diabetes. Frontiers in Pharmacology. 12: 683156
Cohen RA, Murdoch CE, Watanabe Y, et al. (2016) Endothelial Cell Redox Regulation of Ischemic Angiogenesis. Journal of Cardiovascular Pharmacology
Mei Y, Thompson MD, Cohen RA, et al. (2015) Autophagy and oxidative stress in cardiovascular diseases. Biochimica Et Biophysica Acta. 1852: 243-51
Qin Z, Hou X, Weisbrod RM, et al. (2014) Nox2 mediates high fat high sucrose diet-induced nitric oxide dysfunction and inflammation in aortic smooth muscle cells. Journal of Molecular and Cellular Cardiology. 72: 56-63
Qin F, Siwik DA, Lancel S, et al. (2013) Hydrogen peroxide-mediated SERCA cysteine 674 oxidation contributes to impaired cardiac myocyte relaxation in senescent mouse heart. Journal of the American Heart Association. 2: e000184
Lancel S, Qin F, Lennon SL, et al. (2010) Oxidative posttranslational modifications mediate decreased SERCA activity and myocyte dysfunction in Galphaq-overexpressing mice. Circulation Research. 107: 228-32
Tong X, Evangelista A, Cohen RA. (2010) Targeting the redox regulation of SERCA in vascular physiology and disease. Current Opinion in Pharmacology. 10: 133-8
Lancel S, Zhang J, Evangelista A, et al. (2009) Nitroxyl activates SERCA in cardiac myocytes via glutathiolation of cysteine 674. Circulation Research. 104: 720-3
Malester B, Tong X, Ghiu I, et al. (2007) Transgenic expression of a dominant negative K(ATP) channel subunit in the mouse endothelium: effects on coronary flow and endothelin-1 secretion. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 21: 2162-72
Tong X, Porter LM, Liu G, et al. (2006) Consequences of cardiac myocyte-specific ablation of KATP channels in transgenic mice expressing dominant negative Kir6 subunits. American Journal of Physiology. Heart and Circulatory Physiology. 291: H543-51
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