Belma Turan
Affiliations: | Biophysics | Ankara University |
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Aktay I, Billur D, Tuncay E, et al. (2023) An Overexpression of SLC30A6 Gene Contributes to Cardiomyocyte Dysfunction via Affecting Mitochondria and Inducing Activations in K-Acetylation and Epigenetic Proteins. Biochemical Genetics |
Billur D, Olgar Y, Durak A, et al. (2023) An increase in intercellular crosstalk and electrotonic coupling between cardiomyocytes and nonmyocytes reshapes the electrical conduction in the metabolic heart characterized by short QT intervals in ECGs. Cell Biochemistry and Function. 41: 1526-1542 |
Tuncay E, Aktay I, Turan B. (2023) Overexpression of Slc30a7/ZnT7 increases the mitochondrial matrix levels of labile Zn and modifies histone modification in hyperinsulinemic cardiomyoblasts. Journal of Trace Elements in Medicine and Biology : Organ of the Society For Minerals and Trace Elements (Gms). 78: 127198 |
Aktay I, Bitirim CV, Olgar Y, et al. (2023) Cardioprotective role of a magnolol and honokiol complex in the prevention of doxorubicin-mediated cardiotoxicity in adult rats. Molecular and Cellular Biochemistry |
Tuncay E, Gando I, Huo JY, et al. (2023) The cardioprotective role of sirtuins is mediated in part by regulating K channel surface expression. American Journal of Physiology. Cell Physiology |
Durak A, Turan B. (2022) Liraglutide provides cardioprotection through the recovery of mitochondrial dysfunction and oxidative stress in aging hearts. Journal of Physiology and Biochemistry |
Turan B, Durak A, Olgar Y, et al. (2022) Comparisons of pleiotropic effects of SGLT2 inhibition and GLP-1 agonism on cardiac glucose intolerance in heart dysfunction. Molecular and Cellular Biochemistry |
Bitirim CV, Ozer ZB, Aydos D, et al. (2022) Cardioprotective effect of extracellular vesicles derived from ticagrelor-pretreated cardiomyocyte on hyperglycemic cardiomyocytes through alleviation of oxidative and endoplasmic reticulum stress. Scientific Reports. 12: 5651 |
Durak A, Olgar Y, Genc K, et al. (2022) STIM1-Orai1 interaction mediated calcium influx activation contributes to cardiac contractility of insulin-resistant rats. Bmc Cardiovascular Disorders. 22: 147 |
Olgar Y, Durak A, Bitirim CV, et al. (2021) Insulin acts as an atypical KCNQ1/KCNE1-current activator and reverses long QT in insulin-resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β -adrenergic receptor signaling pathway. Journal of Cellular Physiology |