Volodymyr Dzhala, Ph.D.
Affiliations: | Neurology | Massachusetts General Hospital & Harvard Medical School, Boston, MA, United States |
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"Volodymyr Dzhala"Mean distance: 15.33 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentor| Yehezkel Ben-Ari | post-doc | 1997-2001 | INSERM U29/INMED | |
| Kevin Staley | post-doc | 2001-2004 | University of Colorado, Denver | |
| Kevin Staley | research scientist | 2007- | Massachusetts General Hospital & Harvard Medical School | |
| (Instructor) | ||||
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Publications
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| Dzhala V, Fowler AJ, DiMarzio BA, et al. (2022) Analysis of brain region-specific mRNA synthesis and stability by utilizing adult mouse brain slice culture. Star Protocols. 3: 101349 |
| Rahmati N, Normoyle KP, Glykys J, et al. (2021) Unique actions of GABA arising from cytoplasmic chloride microdomains. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Dzhala VI, Staley KJ. (2020) KCC2 chloride transport contributes to the termination of ictal epileptiform activity. Eneuro |
| Blauwblomme T, Dzhala V, Staley K. (2018) Transient ischemia facilitates neuronal chloride accumulation and severity of seizures. Annals of Clinical and Translational Neurology. 5: 1048-1061 |
| Glykys J, Dzhala V, Egawa K, et al. (2017) Chloride Dysregulation, Seizures, and Cerebral Edema: A Relationship with Therapeutic Potential. Trends in Neurosciences |
| Park KI, Dzhala V, Saponjian Y, et al. (2015) What Elements of the Inflammatory System Are Necessary for Epileptogenesis In Vitro?(1,2). Eneuro. 2 |
| Dzhala V, Staley KJ. (2015) Acute and chronic efficacy of bumetanide in an in vitro model of posttraumatic epileptogenesis. Cns Neuroscience & Therapeutics. 21: 173-80 |
| Glykys J, Dzhala V, Egawa K, et al. (2014) Response to comments on "Local impermeant anions establish the neuronal chloride concentration". Science (New York, N.Y.). 345: 1130 |
| Glykys J, Dzhala V, Egawa K, et al. (2014) Local impermeant anions establish the neuronal chloride concentration. Science (New York, N.Y.). 343: 670-5 |
| Polydoro M, Dzhala VI, Pooler AM, et al. (2014) Soluble pathological tau in the entorhinal cortex leads to presynaptic deficits in an early Alzheimer's disease model. Acta Neuropathologica. 127: 257-70 |