Howard L. Weiner

Affiliations: 
Neuroimmunology Brigham and Women's Hospital, Harvard Medical School, Boston, MA, United States 
Website:
http://weinerlab.bwh.harvard.edu/members/WeinerLab_Weiner.html
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"Howard L. Weiner"
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Publications

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Weiner HL. (2024) Immune mechanisms and shared immune targets in neurodegenerative diseases. Nature Reviews. Neurology
Zeng L, Atlas K, Lama T, et al. (2024) GWAS highlights the neuronal contribution to multiple sclerosis susceptibility. Medrxiv : the Preprint Server For Health Sciences
McGinnis JP, Ortiz-Guzman J, Guevara MC, et al. (2024) Common AAV gene therapy vectors show indiscriminate transduction of living human brain cell types. Biorxiv : the Preprint Server For Biology
Teresa Hidalgo E, Grin EA, Tanweer O, et al. (2024) The Pipeline embolization device for the treatment of intracranial aneurysms in pediatric patients with tuberous sclerosis complex: illustrative cases. Journal of Neurosurgery. Case Lessons. 8
Kaul M, Mukherjee D, Weiner HL, et al. (2024) Gut microbiota immune cross-talk in amyotrophic lateral sclerosis. Neurotherapeutics : the Journal of the American Society For Experimental Neurotherapeutics. 21: e00469
Mangani D, Subramanian A, Huang L, et al. (2024) Transcription factor TCF1 binds to RORγt and orchestrates a regulatory network that determines homeostatic Th17 cell state. Immunity
Madill E, Healy BC, Molazadeh N, et al. (2024) Serum glial fibrillary acidic protein predicts disease progression in multiple sclerosis. Annals of Clinical and Translational Neurology
Lin Q, Dorsett Y, Mirza A, et al. (2024) Meta-analysis identifies common gut microbiota associated with multiple sclerosis. Genome Medicine. 16: 94
Izzy S, Yahya T, Albastaki O, et al. (2024) High-salt diet induces microbiome dysregulation, neuroinflammation and anxiety in the chronic period after mild repetitive closed head injury in adolescent mice. Brain Communications. 6: fcae147
Rosenzweig N, Kleemann KL, Rust T, et al. (2024) Sex-dependent APOE4 neutrophil-microglia interactions drive cognitive impairment in Alzheimer's disease. Nature Medicine
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