Bradley K. Taylor

Affiliations: 
Anesthesiology and Perioperative Medicine University of Pittsburgh, Pittsburgh, PA, United States 
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Publications

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Cooper AH, Brightwell JJ, Hedden NS, et al. (2018) The left central nucleus of the amygdala contributes to mechanical allodynia and hyperalgesia following right-sided peripheral nerve injury. Neuroscience Letters
Kaushal R, Taylor BK, Jamal AB, et al. (2016) GABA-A receptor activity in the noradrenergic locus coeruleus drives trigeminal neuropathic pain in the rat; contribution of NAα1 receptors in the medial prefrontal cortex. Neuroscience
Rau KK, Hill CE, Harrison BJ, et al. (2016) Cutaneous tissue damage induces long-lasting nociceptive sensitization and regulation of cellular stress- and nerve injury-associated genes in sensory neurons. Experimental Neurology
Griggs RB, Donahue RR, Adkins BG, et al. (2015) Pioglitazone inhibits the development of hyperalgesia and sensitization of spinal nociresponsive neurons in type 2 diabetes. The Journal of Pain : Official Journal of the American Pain Society
Griggs RB, Bardo MT, Taylor BK. (2015) Gabapentin alleviates affective pain after traumatic nerve injury. Neuroreport. 26: 522-7
Fehrenbacher JC, Loverme J, Clarke W, et al. (2009) Rapid pain modulation with nuclear receptor ligands. Brain Research Reviews. 60: 114-24
Guo N, Garcia MM, Taylor BK, et al. (2008) Blockade of micro-opioid receptors in the medial thalamus inhibits acquisition, but not expression, of morphine-induced conditioned place preference. Neuroscience. 151: 948-54
Taylor BK, Abhyankar SS, Vo NT, et al. (2007) Neuropeptide Y acts at Y1 receptors in the rostral ventral medulla to inhibit neuropathic pain. Pain. 131: 83-95
Bivalacqua TJ, Deng W, Kendirci M, et al. (2007) Mesenchymal stem cells alone or ex vivo gene modified with endothelial nitric oxide synthase reverse age-associated erectile dysfunction. American Journal of Physiology. Heart and Circulatory Physiology. 292: H1278-90
Baber SR, Deng W, Master RG, et al. (2007) Intratracheal mesenchymal stem cell administration attenuates monocrotaline-induced pulmonary hypertension and endothelial dysfunction. American Journal of Physiology. Heart and Circulatory Physiology. 292: H1120-8
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