Maury Cole

2006-2017 Neuroscience Scripps Research Institute, La Jolla, La Jolla, CA, United States 
"Maury Cole"
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Montanari C, Kelley LK, Kerr TM, et al. (2019) Nicotine e-cigarette vapor inhalation effects on nicotine & cotinine plasma levels and somatic withdrawal signs in adult male Wistar rats. Psychopharmacology
Vendruscolo JC, Tunstall BJ, Carmack SA, et al. (2017) Compulsive-Like Sufentanil Vapor Self-Administration in the Rat. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology
Kimbrough A, Kim S, Cole M, et al. (2017) Intermittent Access to Ethanol Drinking Facilitates the Transition to Excessive Drinking After Chronic Intermittent Ethanol Vapor Exposure. Alcoholism, Clinical and Experimental Research
de Guglielmo G, Kallupi M, Cole MD, et al. (2017) Voluntary induction and maintenance of alcohol dependence in rats using alcohol vapor self-administration. Psychopharmacology
de Guglielmo G, Crawford E, Kim S, et al. (2016) Recruitment of a Neuronal Ensemble in the Central Nucleus of the Amygdala Is Required for Alcohol Dependence. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 9446-53
George O, Grieder TE, Cole M, et al. (2010) Exposure to chronic intermittent nicotine vapor induces nicotine dependence. Pharmacology, Biochemistry, and Behavior. 96: 104-7
Gilpin NW, Smith AD, Cole M, et al. (2009) Operant behavior and alcohol levels in blood and brain of alcohol-dependent rats. Alcoholism, Clinical and Experimental Research. 33: 2113-23
Gilpin NW, Richardson HN, Cole M, et al. (2008) Vapor inhalation of alcohol in rats. Current Protocols in Neuroscience / Editorial Board, Jacqueline N. Crawley ... [Et Al.]. Unit 9.29
Chu K, Koob GF, Cole M, et al. (2007) Dependence-induced increases in ethanol self-administration in mice are blocked by the CRF1 receptor antagonist antalarmin and by CRF1 receptor knockout. Pharmacology, Biochemistry, and Behavior. 86: 813-21
Finn DA, Snelling C, Fretwell AM, et al. (2007) Increased drinking during withdrawal from intermittent ethanol exposure is blocked by the CRF receptor antagonist D-Phe-CRF(12-41). Alcoholism, Clinical and Experimental Research. 31: 939-49
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