Chihiro Sato
Affiliations: | Washington University School of Medicine, St. Louis, MO, United States |
Area:
NeuroscienceGoogle:
"Chihiro Sato"Mean distance: (not calculated yet)
Parents
Sign in to add mentorTakeshi Iwatsubo | grad student | ||
Randall Bateman | post-doc | ||
Raphael Kopan | post-doc |
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Publications
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Sun Z, Kwon JS, Ren Y, et al. (2024) Modeling late-onset Alzheimer's disease neuropathology via direct neuronal reprogramming. Science (New York, N.Y.). 385: adl2992 |
Horie K, Salvadó G, Barthélemy NR, et al. (2023) CSF MTBR-tau243 is a specific biomarker of tau tangle pathology in Alzheimer's disease. Nature Medicine |
Sun Z, Kwon JS, Ren Y, et al. (2023) Endogenous recapitulation of Alzheimer's disease neuropathology through human 3D direct neuronal reprogramming. Biorxiv : the Preprint Server For Biology |
Barthélemy NR, Saef B, Li Y, et al. (2023) CSF tau phosphorylation occupancies at T217 and T205 represent improved biomarkers of amyloid and tau pathology in Alzheimer's disease. Nature Aging. 3: 391-401 |
Horie K, Li Y, Barthélemy NR, et al. (2023) Change in cerebrospinal fluid tau microtubule binding region detects symptom onset, cognitive decline, tangles, and atrophy in Dominantly Inherited Alzheimer's Disease. Annals of Neurology |
Mahali S, Martinez R, King M, et al. (2022) Defective proteostasis in induced pluripotent stem cell models of frontotemporal lobar degeneration. Translational Psychiatry. 12: 508 |
Horie K, Barthélemy NR, Spina S, et al. (2022) CSF tau microtubule-binding region identifies pathological changes in primary tauopathies. Nature Medicine |
Capano LS, Sato C, Ficulle E, et al. (2022) Recapitulation of endogenous 4R tau expression and formation of insoluble tau in directly reprogrammed human neurons. Cell Stem Cell. 29: 918-932.e8 |
Lim YY, Maruff P, Barthélemy NR, et al. (2022) Association of BDNF Val66Met With Tau Hyperphosphorylation and Cognition in Dominantly Inherited Alzheimer Disease. Jama Neurology |
Ezerskiy LA, Schoch KM, Sato C, et al. (2021) Astrocytic 4R tau expression drives astrocyte reactivity and dysfunction. Jci Insight |