Marina Angelini
Affiliations: | 2014- | Dept. of Anesthesiology-Div.of Mol. Medicine | University of California, Los Angeles, Los Angeles, CA |
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| Nilsson M, Wang K, Mínguez-Viñas T, et al. (2024) Voltage-dependent G-protein regulation of Ca2.2 (N-type) channels. Science Advances. 10: eadp6665 |
| Nilsson M, Wang K, Mínguez-Viñas T, et al. (2024) Electrical and G-protein Regulation of CaV2.2 (N-type) Channels. Biorxiv : the Preprint Server For Biology |
| Fischer MA, Arrieta A, Angelini M, et al. (2024) Decreased Left Atrial Cardiomyocyte Fibroblast Growth Factor 13 Expression Increases Vulnerability to Postoperative Atrial Fibrillation in Humans. Journal of the American Heart Association. 13: e034896 |
| Scranton K, John S, Angelini M, et al. (2024) Cardiac function is regulated by the sodium-dependent inhibition of the sodium-calcium exchanger NCX1. Nature Communications. 15: 3831 |
| Fischer MA, Arrieta A, Angelini M, et al. (2024) Decreased Left Atrial Cardiomyocyte FGF13 Expression Increases Vulnerability to Postoperative Atrial Fibrillation in Humans. Biorxiv : the Preprint Server For Biology |
| Wang Y, Li Q, Tao B, et al. (2023) Fibroblasts in heart scar tissue directly regulate cardiac excitability and arrhythmogenesis. Science (New York, N.Y.). 381: 1480-1487 |
| Nilsson M, Lindström SH, Kaneko M, et al. (2022) An epilepsy-associated K1.2 charge-transfer-center mutation impairs K1.2 and K1.4 trafficking. Proceedings of the National Academy of Sciences of the United States of America. 119: e2113675119 |
| Angelini M, Pezhouman A, Savalli N, et al. (2021) Suppression of ventricular arrhythmias by targeting late L-type Ca2+ current. The Journal of General Physiology. 153 |
| Savalli N, Angelini M, Steccanella F, et al. (2021) The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation. The Journal of General Physiology. 153 |
| Pantazis A, Kaneko M, Angelini M, et al. (2020) Tracking the motion of K 1.2 voltage sensor reveals the molecular perturbations caused by a de novo mutation in a case of epilepsy. The Journal of Physiology |