Marina Angelini

Affiliations: 
2014- Dept. of Anesthesiology-Div.of Mol. Medicine University of California, Los Angeles, Los Angeles, CA 
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"Marina Angelini"
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Nilsson M, Wang K, Mínguez-Viñas T, et al. (2024) Electrical and G-protein Regulation of CaV2.2 (N-type) Channels. Biorxiv : the Preprint Server For Biology
Fischer MA, Arrieta A, Angelini M, et al. (2024) Decreased Left Atrial Cardiomyocyte Fibroblast Growth Factor 13 Expression Increases Vulnerability to Postoperative Atrial Fibrillation in Humans. Journal of the American Heart Association. 13: e034896
Scranton K, John S, Angelini M, et al. (2024) Cardiac function is regulated by the sodium-dependent inhibition of the sodium-calcium exchanger NCX1. Nature Communications. 15: 3831
Fischer MA, Arrieta A, Angelini M, et al. (2024) Decreased Left Atrial Cardiomyocyte FGF13 Expression Increases Vulnerability to Postoperative Atrial Fibrillation in Humans. Biorxiv : the Preprint Server For Biology
Wang Y, Li Q, Tao B, et al. (2023) Fibroblasts in heart scar tissue directly regulate cardiac excitability and arrhythmogenesis. Science (New York, N.Y.). 381: 1480-1487
Nilsson M, Lindström SH, Kaneko M, et al. (2022) An epilepsy-associated K1.2 charge-transfer-center mutation impairs K1.2 and K1.4 trafficking. Proceedings of the National Academy of Sciences of the United States of America. 119: e2113675119
Angelini M, Pezhouman A, Savalli N, et al. (2021) Suppression of ventricular arrhythmias by targeting late L-type Ca2+ current. The Journal of General Physiology. 153
Savalli N, Angelini M, Steccanella F, et al. (2021) The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation. The Journal of General Physiology. 153
Pantazis A, Kaneko M, Angelini M, et al. (2020) Tracking the motion of K 1.2 voltage sensor reveals the molecular perturbations caused by a de novo mutation in a case of epilepsy. The Journal of Physiology
Savalli N, Angelini M, Steccanella F, et al. (2020) The Contribution of the Individual Voltage Sensors to the Activation of Skeletal CaV1.1 Channels Biophysical Journal. 118: 105a
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