Jakob Hartmann

2009-2014 MPIP Munich, München, Bayern, Germany 
"Jakob Hartmann"
Mean distance: (not calculated yet)
BETA: Related publications


You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Hartmann J, Bajaj T, Klengel C, et al. (2021) Mineralocorticoid receptors dampen glucocorticoid receptor sensitivity to stress via regulation of FKBP5. Cell Reports. 35: 109185
McCullough KM, Missig G, Robble MA, et al. (2021) Nucleus Accumbens Medium Spiny Neuron Subtypes Differentially Regulate Stress-Associated Alterations in Sleep Architecture. Biological Psychiatry
McCullough KM, Chatzinakos C, Hartmann J, et al. (2020) Author Correction: Genome-wide translational profiling of amygdala Crh-expressing neurons reveals role for CREB in fear extinction learning. Nature Communications. 11: 5529
Huckins LM, Chatzinakos C, Breen MS, et al. (2020) Analysis of Genetically Regulated Gene Expression Identifies a Prefrontal PTSD Gene, SNRNP35, Specific to Military Cohorts. Cell Reports. 31: 107716
Dedic N, Kühne C, Gomes KS, et al. (2019) Deletion of CRH From GABAergic Forebrain Neurons Promotes Stress Resilience and Dampens Stress-Induced Changes in Neuronal Activity. Frontiers in Neuroscience. 13: 986
van Weert LTCM, Buurstede JC, Sips HCM, et al. (2019) Identification of mineralocorticoid receptor target genes in the mouse hippocampus. Journal of Neuroendocrinology. e12735
Maddox SA, Hartmann J, Ross RA, et al. (2019) Deconstructing the Gestalt: Mechanisms of Fear, Threat, and Trauma Memory Encoding. Neuron. 102: 60-74
Dedic N, Kühne C, Jakovcevski M, et al. (2018) Chronic CRH depletion from GABAergic, long-range projection neurons in the extended amygdala reduces dopamine release and increases anxiety. Nature Neuroscience
McCullough KM, Morrison FG, Hartmann J, et al. (2018) Quantified Coexpression Analysis of Central Amygdala Subpopulations. Eneuro. 5
Dedic N, Pöhlmann ML, Richter JS, et al. (2017) Cross-disorder risk gene CACNA1C differentially modulates susceptibility to psychiatric disorders during development and adulthood. Molecular Psychiatry
See more...