Alfred Goldberg
Affiliations: | Harvard Medical School, Boston, MA, United States |
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Children
Sign in to add traineeJudith Frydman | grad student | (Neurotree) | |
Ami Navon | post-doc | Harvard Medical School | |
Xiaobo Qiu | post-doc | Harvard Medical School (Neurotree) |
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Publications
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Lee D, Zhu Y, Colson L, et al. (2023) Molecular mechanism for activation of the 26S proteasome by ZFAND5. Molecular Cell. 83: 2959-2975.e7 |
Zhou M, Fang R, Colson L, et al. (2023) HUWE1 Amplifies Ubiquitin Modifications to Broadly Stimulate Clearance of Proteins and Aggregates. Biorxiv : the Preprint Server For Biology |
Lee D, Zhu Y, Colson L, et al. (2023) Molecular mechanisms for activation of the 26S proteasome. Biorxiv : the Preprint Server For Biology |
Lee D, Goldberg AL. (2022) 26S proteasomes become stably activated upon heat shock when ubiquitination and protein degradation increase. Proceedings of the National Academy of Sciences of the United States of America. 119: e2122482119 |
Collins GA, Sha Z, Kuo CL, et al. (2022) Mammalian Ddi2 is a shuttling factor containing a retroviral protease domain that influences binding of ubiquitylated proteins and proteasomal degradation. The Journal of Biological Chemistry. 101875 |
Goldberg AL, Kim HT, Lee D, et al. (2021) Mechanisms That Activate 26S Proteasomes and Enhance Protein Degradation. Biomolecules. 11 |
VerPlank JJS, Tyrkalska SD, Fleming A, et al. (2020) cGMP via PKG activates 26S proteasomes and enhances degradation of proteins, including ones that cause neurodegenerative diseases. Proceedings of the National Academy of Sciences of the United States of America. 117: 14220-14230 |
Collins GA, Goldberg AL. (2020) Proteins containing ubiquitin-like (Ubl) domains not only bind to 26S proteasomes but also induce their activation. Proceedings of the National Academy of Sciences of the United States of America |
Sha Z, Blyszcz T, González-Prieto R, et al. (2019) Inhibiting ubiquitination causes an accumulation of SUMOylated newly synthesized nuclear proteins at PML bodies. The Journal of Biological Chemistry |
Jiang TX, Zou JB, Zhu QQ, et al. (2019) SIP/CacyBP promotes autophagy by regulating levels of BRUCE/Apollon, which stimulates LC3-I degradation. Proceedings of the National Academy of Sciences of the United States of America |