Kelvin Luk

McGill University, Montreal, QC, Canada 
"Kelvin Luk"
Mean distance: (not calculated yet)
BETA: Related publications


You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Boyd SL, Kuhn NC, Patterson JR, et al. (2023) Developmental exposure to the Parkinson's disease-associated organochlorine pesticide dieldrin alters dopamine neurotransmission in α-synuclein pre-formed fibril (PFF)-injected mice. Toxicological Sciences : An Official Journal of the Society of Toxicology
Steinkellner T, Conrad WS, Kovacs I, et al. (2021) Dopamine neurons exhibit emergent glutamatergic identity in Parkinson's disease. Brain : a Journal of Neurology
Wu Q, Shaikh MA, Meymand ES, et al. (2020) Neuronal activity modulates alpha-synuclein aggregation and spreading in organotypic brain slice cultures and in vivo. Acta Neuropathologica
Chmielarz P, Er Ş, Konovalova J, et al. (2020) GDNF/RET Signaling Pathway Activation Eliminates Lewy Body Pathology in Midbrain Dopamine Neurons. Movement Disorders : Official Journal of the Movement Disorder Society
Hoban DB, Shrigley S, Mattsson B, et al. (2020) Impact of α-synuclein pathology on transplanted hESC-derived dopaminergic neurons in a humanized α-synuclein rat model of PD. Proceedings of the National Academy of Sciences of the United States of America
Wong YC, Luk K, Purtell K, et al. (2019) Neuronal vulnerability in Parkinson disease: Should the focus be on axons and synaptic terminals? Movement Disorders : Official Journal of the Movement Disorder Society
Luk KC. (2019) Oxidative stress and α-synuclein conspire in vulnerable neurons to promote Parkinson's disease progression. The Journal of Clinical Investigation. 130
Luna E, Decker SC, Riddle DM, et al. (2018) Differential α-synuclein expression contributes to selective vulnerability of hippocampal neuron subpopulations to fibril-induced toxicity. Acta Neuropathologica
Tapias V, Hu X, Luk KC, et al. (2017) Synthetic alpha-synuclein fibrils cause mitochondrial impairment and selective dopamine neurodegeneration in part via iNOS-mediated nitric oxide production. Cellular and Molecular Life Sciences : Cmls
Osterberg VR, Spinelli KJ, Weston LJ, et al. (2015) Progressive aggregation of alpha-synuclein and selective degeneration of lewy inclusion-bearing neurons in a mouse model of parkinsonism. Cell Reports. 10: 1252-60
See more...