Andrew Paul Tosolini

Affiliations: 
University College London, London, United Kingdom 
Area:
Motor neurons; ALS; MND; tracing; axonal transport
Google:
"Andrew Tosolini"
Mean distance: (not calculated yet)
 
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Sleigh JN, Villarroel-Campos D, Surana S, et al. (2023) Boosting peripheral BDNF rescues impaired in vivo axonal transport in CMT2D mice. Jci Insight
Negro S, Lauria F, Stazi M, et al. (2022) Hydrogen peroxide induced by nerve injury promotes axon regeneration via connective tissue growth factor Acta Neuropathologica Communications
Tosolini AP, Sleigh JN, Surana S, et al. (2022) BDNF-dependent modulation of axonal transport is selectively impaired in ALS. Acta Neuropathologica Communications. 10: 121
Rhymes ER, Tosolini AP, Fellows AD, et al. (2022) Bimodal regulation of axonal transport by the GDNF-RET signalling axis in healthy and diseased motor neurons. Cell Death & Disease. 13: 584
Tosolini AP, Villarroel-Campos D, Schiavo G, et al. (2021) Expanding the Toolkit for In Vivo Imaging of Axonal Transport. Journal of Visualized Experiments : Jove
Tosolini AP, Mentis GZ, Martin JH. (2021) Editorial: Dysfunction and Repair of Neural Circuits for Motor Control. Frontiers in Molecular Neuroscience. 14: 669824
Tosolini AP, Sleigh JN. (2020) Intramuscular Delivery of Gene Therapy for Targeting the Nervous System. Frontiers in Molecular Neuroscience. 13: 129
Sleigh JN, Tosolini AP, Schiavo G. (2020) In Vivo Imaging of Anterograde and Retrograde Axonal Transport in Rodent Peripheral Nerves. Methods in Molecular Biology (Clifton, N.J.). 2143: 271-292
Sleigh JN, Tosolini AP, Gordon D, et al. (2020) Mice Carrying ALS Mutant TDP-43, but Not Mutant FUS, Display In Vivo Defects in Axonal Transport of Signaling Endosomes. Cell Reports. 30: 3655-3662.e2
Fratta P, Birsa N, Tosolini AP, et al. (2019) Travelling Together: A Unifying Pathomechanism for ALS. Trends in Neurosciences
See more...