Gabriel L. McKinsey
Affiliations: | University of California, San Francisco, San Francisco, CA |
Area:
Developmental NeuroscienceGoogle:
"Gabriel McKinsey"Mean distance: 14.52 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentor| John L. R. Rubenstein | grad student | UCSF | |
| Nirao M. Shah | post-doc | 2014- | UCSF |
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Publications
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| Su-Feher L, Rubin AN, Silberberg SN, et al. (2022) Single cell enhancer activity distinguishes GABAergic and cholinergic lineages in embryonic mouse basal ganglia. Proceedings of the National Academy of Sciences of the United States of America. 119: e2108760119 |
| McKinsey GL, Lizama CO, Keown-Lang AE, et al. (2020) A new genetic strategy for targeting microglia in development and disease. Elife. 9 |
| Santander N, Lizama CO, Meky E, et al. (2020) Lack of Flvcr2 impairs brain angiogenesis without affecting the blood-brain barrier. The Journal of Clinical Investigation |
| McGregor MM, McKinsey GL, Girasole AE, et al. (2019) Functionally Distinct Connectivity of Developmentally Targeted Striosome Neurons. Cell Reports. 29: 1419-1428.e5 |
| Lindtner S, Catta-Preta R, Tian H, et al. (2019) Genomic Resolution of DLX-Orchestrated Transcriptional Circuits Driving Development of Forebrain GABAergic Neurons. Cell Reports. 28: 2048-2063.e8 |
| Pai EL, Vogt D, Clemente-Perez A, et al. (2019) Mafb and c-Maf Have Prenatal Compensatory and Postnatal Antagonistic Roles in Cortical Interneuron Fate and Function. Cell Reports. 26: 1157-1173.e5 |
| McKinsey G, Ahmed OM, Shah NM. (2018) Neural control of sexually dimorphic social behaviors. Current Opinion in Physiology. 6: 89-95 |
| Silberberg SN, Taher L, Lindtner S, et al. (2016) Subpallial Enhancer Transgenic Lines: a Data and Tool Resource to Study Transcriptional Regulation of GABAergic Cell Fate. Neuron. 92: 59-74 |
| Haliburton GD, McKinsey GL, Pollard KS. (2015) Disruptions in a cluster of computationally identified enhancers near FOXC1 and GMDS may influence brain development. Neurogenetics |
| Silbereis JC, Nobuta H, Tsai HH, et al. (2014) Olig1 function is required to repress dlx1/2 and interneuron production in Mammalian brain. Neuron. 81: 574-87 |