Iain L. Campbell

Affiliations: 
Neuropharmacology Scripps Research Institute, La Jolla, La Jolla, CA, United States 
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"Iain Campbell"
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West PK, Viengkhou B, Campbell IL, et al. (2022) Microglia shield the murine brain from damage mediated by the cytokines IL-6 and IFN-α. Frontiers in Immunology. 13: 1036799
West PK, McCorkindale AN, Guennewig B, et al. (2022) The cytokines interleukin-6 and interferon-α induce distinct microglia phenotypes. Journal of Neuroinflammation. 19: 96
Jung SR, Ashhurst TM, West PK, et al. (2020) Contribution of STAT1 to innate and adaptive immunity during type I interferon-mediated lethal virus infection. Plos Pathogens. 16: e1008525
Nitsch L, Zimmermann J, Krauthausen M, et al. (2019) CNS-Specific Synthesis of Interleukin 23 Induces a Progressive Cerebellar Ataxia and the Accumulation of Both T and B Cells in the Brain: Characterization of a Novel Transgenic Mouse Model. Molecular Neurobiology
West PK, Viengkhou B, Campbell IL, et al. (2019) Microglia responses to interleukin-6 and type I interferons in neuroinflammatory disease. Glia. 67: 1821-1841
Li W, Viengkhou B, Denyer G, et al. (2018) Microglia have a more extensive and divergent response to interferon-α compared with astrocytes. Glia
Huber M, Suprunenko T, Ashhurst T, et al. (2017) IRF9 prevents CD8(+) T cell exhaustion in an extrinsic manner during acute LCMV infection. Journal of Virology
Petković F, Campbell IL, Gonzalez B, et al. (2017) Reduced cuprizone-induced cerebellar demyelination in mice with astrocyte-targeted production of IL-6 is associated with chronically activated, but less responsive microglia. Journal of Neuroimmunology. 310: 97-102
Li W, Hofer MJ, Songkhunawej P, et al. (2017) Type I Interferon-Regulated Gene Expression and Signaling in Murine Mixed Glial Cells Lacking Signal Transducers and Activators of Transcription 1 or 2 or Interferon Regulatory Factor 9. The Journal of Biological Chemistry
Zimmermann J, Emrich M, Krauthausen M, et al. (2017) IL-17A Promotes Granulocyte Infiltration, Myelin Loss, Microglia Activation, and Behavioral Deficits During Cuprizone-Induced Demyelination. Molecular Neurobiology
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