Bilal Malik
Affiliations: | Institute of Neurology | University College London |
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| Elkaddi N, Malik B, Spielman AI, et al. (2022) Reprogramming cultured human fungiform (HBO) taste cells into neuron-like cells through in vitro induction. In Vitro Cellular & Developmental Biology. Animal |
| Malik BR, Maddison DC, Smith GA, et al. (2019) Autophagic and endo-lysosomal dysfunction in neurodegenerative disease. Molecular Brain. 12: 100 |
| Higham JP, Malik BR, Buhl E, et al. (2019) Alzheimer's Disease Associated Genes Ankyrin and Tau Cause Shortened Lifespan and Memory Loss in . Frontiers in Cellular Neuroscience. 13: 260 |
| Killick R, Ribe EM, Al-Shawi R, et al. (2014) Clusterin regulates β-amyloid toxicity via Dickkopf-1-driven induction of the wnt-PCP-JNK pathway. Molecular Psychiatry. 19: 88-98 |
| Malik B, Fernandes C, Killick R, et al. (2012) Oligomeric amyloid-β peptide affects the expression of genes involved in steroid and lipid metabolism in primary neurons. Neurochemistry International. 61: 321-33 |
| Killick R, Ribe EM, Malik B, et al. (2010) P2-311: Expression of an Aβ transcription signature gene set in mouse and human brain Alzheimer's & Dementia. 6: S404-S405 |
| Malik B, Currais A, Soriano S. (2008) Cell cycle-driven neuronal apoptosis specifically linked to amyloid peptide Abeta1-42 exposure is not exacerbated in a mouse model of presenilin-1 familial Alzheimer's disease. Journal of Neurochemistry. 106: 912-6 |
| Malik B, Currais A, Andres A, et al. (2008) Loss of neuronal cell cycle control as a mechanism of neurodegeneration in the presenilin-1 Alzheimer's disease brain. Cell Cycle (Georgetown, Tex.). 7: 637-46 |
| Currais A, Malik B, Noble W, et al. (2006) AbstractPoster presentation: Tuesday posterP3-410: Analysis of the role of presenilin 1 on tau homeostasis Alzheimers & Dementia. 2 |
| Malik B, Killick R, Ritchie W, et al. (2004) P1-159 Amyloid beta regulation of gene expression in primary mouse neuronal cultures Neurobiology of Aging. 25 |