Giovanna Mulas, PhD

Affiliations: 
Univ of Cagliari 
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"Giovanna Mulas"
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Publications

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Casu MA, Mocci I, Isola R, et al. (2020) Neuroprotection by the Immunomodulatory Drug Pomalidomide in the LRRK2 Genetic Model of Parkinson's Disease. Frontiers in Aging Neuroscience. 12: 31
Boi L, Pisanu A, Greig N, et al. (2019) Immunomodulatory drugs alleviate l-dopa-induced dyskinesia in a rat model of Parkinson's disease. Movement Disorders : Official Journal of the Movement Disorder Society
Devoto P, Flore G, Saba P, et al. (2019) Noradrenergic terminals are the primary source of α-adrenoceptor mediated dopamine release in the medial prefrontal cortex. Progress in Neuro-Psychopharmacology & Biological Psychiatry. 90: 97-103
Cannizzaro C, Talani G, Brancato A, et al. (2018) Dopamine restores limbic memory loss, dendritic spine structure and NMDAR-dependent LTD in the nucleus accumbens of alcohol-withdrawn rats. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Pisanu A, Boi L, Mulas G, et al. (2018) Neuroinflammation in L-DOPA-induced dyskinesia: beyond the immune function. Journal of Neural Transmission (Vienna, Austria : 1996)
Carta AR, Mulas G, Bortolanza M, et al. (2016) L-DOPA-induced dyskinesia and neuroinflammation: do microglia and astrocytes play a role? The European Journal of Neuroscience
Mulas G, Espa E, Fenu S, et al. (2016) Differential induction of dyskinesia and neuroinflammation by pulsatile versus continuous l-DOPA delivery in the 6-OHDA model of Parkinson's disease. Experimental Neurology. 286: 83-92
Castelli MP, Spiga S, Perra A, et al. (2016) α2A adrenergic receptors highly expressed in mesoprefrontal dopamine neurons. Neuroscience
Joers V, Tansey MG, Mulas G, et al. (2016) Microglial phenotypes in Parkinson's disease and animal models of the disease. Progress in Neurobiology
Enrico P, Migliore M, Spiga S, et al. (2016) Morphofunctional alterations in ventral tegmental area dopamine neurons in acute and prolonged opiates withdrawal. A computational perspective. Neuroscience
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