. .
Affiliations: | 2004- | Biological Sciences | Korea Advanced Institute of Science and Technology, Daejeon, South Korea |
Area:
NeuroscienceGoogle:
". ."Mean distance: (not calculated yet)
BETA: Related publications
See more...
Publications
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
Kim D, Cheong E, Shin HS. (2018) Overcoming Depression by Inhibition of Neural Burst Firing. Neuron. 98: 878-879 |
Gangadharan G, Shin J, Kim SW, et al. (2016) Medial septal GABAergic projection neurons promote object exploration behavior and type 2 theta rhythm. Proceedings of the National Academy of Sciences of the United States of America |
Kim MY, Kim HY, Hong J, et al. (2015) CXXC5 plays a role as a transcription activator for myelin genes on oligodendrocyte differentiation. Glia |
Kim Y, Kim SH, Kim KH, et al. (2015) Age-dependent gait abnormalities in mice lacking the Rnf170 gene linked to human autosomal-dominant sensory ataxia. Human Molecular Genetics |
Kyung T, Lee S, Kim JE, et al. (2015) Optogenetic control of endogenous Ca(2+) channels in vivo. Nature Biotechnology |
Han YE, Ryu SY, Park SH, et al. (2015) Ca(2+) clearance by plasmalemmal NCLX, Li(+)-permeable Na (+)/Ca (2+) exchanger, is required for the sustained exocytosis in rat insulinoma INS-1 cells. Pflugers Archiv : European Journal of Physiology |
Jo S, Yarishkin O, Hwang YJ, et al. (2014) GABA from reactive astrocytes impairs memory in mouse models of Alzheimer's disease. Nature Medicine. 20: 886-96 |
Jang JY, Koh YJ, Lee SH, et al. (2013) Conditional ablation of LYVE-1+ cells unveils defensive roles of lymphatic vessels in intestine and lymph nodes. Blood. 122: 2151-61 |
Won H, Lee HR, Gee HY, et al. (2012) Autistic-like social behaviour in Shank2-mutant mice improved by restoring NMDA receptor function. Nature. 486: 261-5 |
Kim J, Woo J, Park YG, et al. (2011) Thalamic T-type Ca²+ channels mediate frontal lobe dysfunctions caused by a hypoxia-like damage in the prefrontal cortex. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 4063-73 |