Jing Wei

Affiliations: 
State University of New York, Buffalo, Buffalo, NY, United States 
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Wei J, Cheng J, Waddell NJ, et al. (2020) DNA Methyltransferase 3A Is Involved in the Sustained Effects of Chronic Stress on Synaptic Functions and Behaviors. Cerebral Cortex (New York, N.Y. : 1991)
Seo JS, Wei J, Qin L, et al. (2019) Correction: Cellular and molecular basis for stress-induced depression. Molecular Psychiatry
Jin J, Bhatti DL, Lee KW, et al. (2019) Ahnak scaffolds p11/Anxa2 complex and L-type voltage-gated calcium channel and modulates depressive behavior. Molecular Psychiatry
Zheng Y, Liu A, Wang ZJ, et al. (2019) Inhibition of EHMT1/2 rescues synaptic and cognitive functions for Alzheimer's disease. Brain : a Journal of Neurology
Qin L, Ma K, Wang ZJ, et al. (2018) Publisher Correction: Social deficits in Shank3-deficient mouse models of autism are rescued by histone deacetylase (HDAC) inhibition. Nature Neuroscience
Qin L, Ma K, Wang ZJ, et al. (2018) Social deficits in Shank3-deficient mouse models of autism are rescued by histone deacetylase (HDAC) inhibition. Nature Neuroscience
Yuen EY, Wei J, Yan Z. (2017) Molecular and Epigenetic Mechanisms for the Complex Effects of Stress on Synaptic Physiology and Cognitive Functions. The International Journal of Neuropsychopharmacology
Wei J, Zhong P, Qin L, et al. (2017) Chemicogenetic Restoration of the Prefrontal Cortex to Amygdala Pathway Ameliorates Stress-Induced Deficits. Cerebral Cortex (New York, N.Y. : 1991). 1-11
Qin L, Liu W, Ma K, et al. (2016) The ADHD-linked human dopamine D4 receptor variant D4.7 induces over-suppression of NMDA receptor function in prefrontal cortex. Neurobiology of Disease
Seo JS, Wei J, Qin L, et al. (2016) Cellular and molecular basis for stress-induced depression. Molecular Psychiatry
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