Luye Qin
Affiliations: | State University of New York, Buffalo, Buffalo, NY, United States |
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| Ma K, Taylor C, Williamson M, et al. (2023) Diminished activity-dependent BDNF signaling differentially causes autism-like behavioral deficits in male and female mice. Frontiers in Psychiatry. 14: 1182472 |
| Williams JB, Cao Q, Wang W, et al. (2023) Inhibition of histone methyltransferase Smyd3 rescues NMDAR and cognitive deficits in a tauopathy mouse model. Nature Communications. 14: 91 |
| Rapanelli M, Williams JB, Ma K, et al. (2022) Targeting histone demethylase LSD1 for treatment of deficits in autism mouse models. Molecular Psychiatry |
| Qin L, Williams JB, Tan T, et al. (2021) Deficiency of autism risk factor ASH1L in prefrontal cortex induces epigenetic aberrations and seizures. Nature Communications. 12: 6589 |
| Qin L, Ma K, Yan Z. (2021) Rescue of histone hypoacetylation and social deficits by ketogenic diet in a Shank3 mouse model of autism. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology |
| Zhong P, Qin L, Yan Z. (2020) Dopamine Differentially Regulates Response Dynamics of Prefrontal Cortical Principal Neurons and Interneurons to Optogenetic Stimulation of Inputs from Ventral Tegmental Area. Cerebral Cortex (New York, N.Y. : 1991) |
| Qin L, Actor-Engel HS, Woo MS, et al. (2019) An Increase of Excitatory-to-Inhibitory Synaptic Balance in the Contralateral Cortico-Striatal Pathway Underlies Improved Stroke Recovery in BDNF Val66Met SNP Mice. Neurorehabilitation and Neural Repair. 1545968319872997 |
| Rapanelli M, Tan T, Wang W, et al. (2019) Behavioral, circuitry, and molecular aberrations by region-specific deficiency of the high-risk autism gene Cul3. Molecular Psychiatry |
| Qin L, Ma K, Yan Z. (2019) Chemogenetic Activation of Prefrontal Cortex in Shank3-Deficient Mice Ameliorates Social Deficits, NMDAR Hypofunction, and Sgk2 Downregulation. Iscience. 17: 24-35 |
| Qin L, Ma K, Wang ZJ, et al. (2018) Publisher Correction: Social deficits in Shank3-deficient mouse models of autism are rescued by histone deacetylase (HDAC) inhibition. Nature Neuroscience |