William Gibson

Affiliations: 
Dana-Farber Cancer Institute and Harvard Medical School, USA 
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"William Gibson"
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Gibson WJ, Sadagopan A, Shoba VM, et al. (2023) Bifunctional Small Molecules That Induce Nuclear Localization and Targeted Transcriptional Regulation. Journal of the American Chemical Society
Gibson WJ, Sadagopan A, Shoba VM, et al. (2023) Bifunctional small molecules that induce nuclear localization and targeted transcriptional regulation. Biorxiv : the Preprint Server For Biology
Nichols CA, Gibson WJ, Brown MS, et al. (2020) Loss of heterozygosity of essential genes represents a widespread class of potential cancer vulnerabilities. Nature Communications. 11: 2517
Holst F, Werner HMJ, Mjøs S, et al. (2018) Amplification Associates with Aggressive Phenotype but Not Markers of AKT-MTOR Signaling in Endometrial Carcinoma. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research
Nichols CA, Paolella BR, Gibson WJ, et al. (2018) Abstract 3003: Loss of heterozygosity of essential genes represents a novel class of cancer vulnerabilities Cancer Research. 78: 3003-3003
Bi WL, Greenwald NF, Abedalthagafi M, et al. (2017) Erratum: Genomic landscape of high-grade meningiomas. Npj Genomic Medicine. 2: 26
Bi WL, Greenwald NF, Abedalthagafi M, et al. (2017) Genomic landscape of high-grade meningiomas. Npj Genomic Medicine. 2
Holst F, Hoivik EA, Gibson WJ, et al. (2017) Corrigendum: Recurrent hormone-binding domain truncated ESR1 amplifications in primary endometrial cancers suggest their implication in hormone independent growth. Scientific Reports. 7: 46873
Paolella BR, Gibson WJ, Urbanski LM, et al. (2017) Copy-number and gene dependency analysis reveals partial copy loss of wild-type SF3B1 as a novel cancer vulnerability. Elife. 6
Paolella BR, Gibson WJ, Urbanski LM, et al. (2017) Author response: Copy-number and gene dependency analysis reveals partial copy loss of wild-type SF3B1 as a novel cancer vulnerability Elife
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