Stacey J. Sukoff Rizzo, Ph.D.

Affiliations: 
The Jackson Laboratory, Bar Harbor, ME, United States 
Area:
Behavioral Neuroscience
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"Stacey Sukoff Rizzo"
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Publications

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Onos KD, Rizzo SJ, Howell GR, et al. (2015) Toward more predictive genetic mouse models of Alzheimer's disease. Brain Research Bulletin
Asinof SK, Sukoff Rizzo SJ, Buckley AR, et al. (2015) Independent Neuronal Origin of Seizures and Behavioral Comorbidities in an Animal Model of a Severe Childhood Genetic Epileptic Encephalopathy. Plos Genetics. 11: e1005347
Gonzales C, Zaleska MM, Riddell DR, et al. (2014) Alternative method of oral administration by peanut butter pellet formulation results in target engagement of BACE1 and attenuation of gavage-induced stress responses in mice. Pharmacology, Biochemistry, and Behavior. 126: 28-35
Sukoff Rizzo SJ, Lotarski SM, Stolyar P, et al. (2014) Behavioral characterization of striatal-enriched protein tyrosine phosphatase (STEP) knockout mice. Genes, Brain, and Behavior. 13: 643-52
Paumier KL, Sukoff Rizzo SJ, Berger Z, et al. (2013) Behavioral characterization of A53T mice reveals early and late stage deficits related to Parkinson's disease. Plos One. 8: e70274
Rizzo SJ, Edgerton JR, Hughes ZA, et al. (2013) Future viable models of psychiatry drug discovery in pharma. Journal of Biomolecular Screening. 18: 509-21
Hughes ZA, Neal SJ, Smith DL, et al. (2013) Negative allosteric modulation of metabotropic glutamate receptor 5 results in broad spectrum activity relevant to treatment resistant depression. Neuropharmacology. 66: 202-14
Sukoff Rizzo SJ, Neal SJ, Hughes ZA, et al. (2012) Evidence for sustained elevation of IL-6 in the CNS as a key contributor of depressive-like phenotypes. Translational Psychiatry. 2: e199
Sukoff Rizzo SJ, Leonard SK, Gilbert A, et al. (2011) The metabotropic glutamate receptor 7 allosteric modulator AMN082: a monoaminergic agent in disguise? The Journal of Pharmacology and Experimental Therapeutics. 338: 345-52
Kelly MP, Logue SF, Brennan J, et al. (2010) Phosphodiesterase 11A in brain is enriched in ventral hippocampus and deletion causes psychiatric disease-related phenotypes. Proceedings of the National Academy of Sciences of the United States of America. 107: 8457-62
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