Valeria Cavalli

Washington University, Saint Louis, St. Louis, MO 
nerve regeneration
"Valeria Cavalli"
Mean distance: 14.96 (cluster 11)
BETA: Related publications


You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Carlin D, Halevi AE, Ewan E, et al. (2019) Nociceptor deletion of Tsc2 enhances axon regeneration by inducing a conditioning injury response in dorsal root ganglia. Eneuro
Deng PY, Carlin D, Mi Oh Y, et al. (2018) Voltage-Independent SK Channel Dysfunction Causes Neuronal Hyperexcitability in the Hippocampus of KO mice. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Carlin D, Golden JP, Mogha A, et al. (2018) Deletion of Tsc2 in Nociceptors Reduces Target Innervation, Ion Channel Expression, and Sensitivity to Heat. Eneuro. 5
Mogha A, Harty BL, Carlin D, et al. (2016) Gpr126/Adgrg6 Has Schwann Cell Autonomous and Nonautonomous Functions in Peripheral Nerve Injury and Repair. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 12351-12367
Cho Y, Sloutsky R, Naegle KM, et al. (2015) Injury-Induced HDAC5 Nuclear Export Is Essential for Axon Regeneration. Cell. 161: 691
Cho Y, Shin JE, Ewan EE, et al. (2015) Activating Injury-Responsive Genes with Hypoxia Enhances Axon Regeneration through Neuronal HIF-1α. Neuron
Cho Y, Park D, Cavalli V. (2015) Filamin A is required in injured axons for HDAC5 activity and axon regeneration. The Journal of Biological Chemistry
Watt D, Dixit R, Cavalli V. (2015) JIP3 Activates Kinesin-1 Motility to Promote Axon Elongation. The Journal of Biological Chemistry. 290: 15512-25
Song W, Cho Y, Watt D, et al. (2015) Tubulin-tyrosine Ligase (TTL)-mediated Increase in Tyrosinated α-Tubulin in Injured Axons Is Required for Retrograde Injury Signaling and Axon Regeneration. The Journal of Biological Chemistry. 290: 14765-75
Myrick LK, Deng PY, Hashimoto H, et al. (2015) Independent role for presynaptic FMRP revealed by an FMR1 missense mutation associated with intellectual disability and seizures. Proceedings of the National Academy of Sciences of the United States of America. 112: 949-56
See more...