Parents
Sign in to add mentor| Dieter Söll | grad student | Yale (Chemistry Tree) | |
| Sandra Schmid | post-doc | 1998-2000 | Scripps Institute |
Children
Sign in to add trainee| Vincent Anh-Quan Pham | research assistant | 2013-2016 | Mass General Hospital and Harvard Medical School (Neurotree) |
| Changkyu Gu | post-doc | Harvard Medical School - Massachusetts General Hospital |
BETA: Related publications
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Publications
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| Zhu K, Mukherjee K, Wei C, et al. (2023) The D2D3 form of uPAR acts as an immunotoxin and may cause diabetes and kidney disease. Science Translational Medicine. 15: eabq6492 |
| Mukherjee K, Gu C, Collins A, et al. (2022) Simultaneous stabilization of actin cytoskeleton in multiple nephron-specific cells protects the kidney from diverse injury. Nature Communications. 13: 2422 |
| Sever S. (2020) Role of actin cytoskeleton in podocytes. Pediatric Nephrology (Berlin, Germany) |
| Hayek SS, Leaf DE, Samman Tahhan A, et al. (2020) Soluble Urokinase Receptor and Acute Kidney Injury. The New England Journal of Medicine. 382: 416-426 |
| Wei C, Li J, Adair BD, et al. (2019) uPAR isoform 2 forms a dimer and induces severe kidney disease in mice. The Journal of Clinical Investigation |
| Sever S, Schiffer M. (2018) Actin dynamics at focal adhesions: a common endpoint and putative therapeutic target for proteinuric kidney diseases. Kidney International |
| Hayek SS, Ko YA, Awad M, et al. (2017) Cardiovascular Disease Biomarkers and suPAR in Predicting Decline in Renal Function: A Prospective Cohort Study. Kidney International Reports. 2: 425-432 |
| Sever S, Gu C. (2017) Podocytes and the actin cytoskeleton as a feasible therapeutic target Periodicum Biologorum. 118: 381-384 |
| Hahm E, Wei C, Fernandez I, et al. (2016) Bone marrow-derived immature myeloid cells are a main source of circulating suPAR contributing to proteinuric kidney disease. Nature Medicine |
| Lee H, Noh H, Mun J, et al. (2016) Anks1a regulates COPII-mediated anterograde transport of receptor tyrosine kinases critical for tumorigenesis. Nature Communications. 7: 12799 |