Yaming Wang
Affiliations: | 2013 | Biology & Biomedical Sciences (Immunology) | Washington University, Saint Louis, St. Louis, MO |
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Sign in to add mentor| Marco Colonna | grad student | 2013 | Washington University | |
| (Disparate Contribution of Endosomal and Cytosolic RNA-Sensing Pathways to Antiviral Immunity.) | ||||
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Publications
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| Ulrich JD, Ulland TK, Mahan TE, et al. (2018) ApoE facilitates the microglial response to amyloid plaque pathology. The Journal of Experimental Medicine |
| Ulland TK, Song WM, Huang SC, et al. (2017) TREM2 Maintains Microglial Metabolic Fitness in Alzheimer's Disease. Cell. 170: 649-663.e13 |
| Yuan P, Condello C, Keene CD, et al. (2016) TREM2 Haplodeficiency in Mice and Humans Impairs the Microglia Barrier Function Leading to Decreased Amyloid Compaction and Severe Axonal Dystrophy. Neuron. 92: 252-264 |
| Song W, Hooli B, Mullin K, et al. (2016) Alzheimer's disease-associated TREM2 variants exhibit either decreased or increased ligand-dependent activation. Alzheimer's & Dementia : the Journal of the Alzheimer's Association |
| Yuan P, Condello C, Keene CD, et al. (2016) TREM2 Haplodeficiency in Mice and Humans Impairs the Microglia Barrier Function Leading to Decreased Amyloid Compaction and Severe Axonal Dystrophy. Neuron. 90: 724-739 |
| Cortez VS, Cervantes-Barragan L, Robinette ML, et al. (2016) Transforming Growth Factor-β Signaling Guides the Differentiation of Innate Lymphoid Cells in Salivary Glands. Immunity |
| Wang Y, Ulland TK, Ulrich JD, et al. (2016) TREM2-mediated early microglial response limits diffusion and toxicity of amyloid plaques. The Journal of Experimental Medicine |
| Ulland TK, Wang Y, Colonna M. (2016) Regulation of microglial survival and proliferation in health and diseases. Seminars in Immunology |
| Colonna M, Wang Y. (2016) TREM2 variants: new keys to decipher Alzheimer disease pathogenesis. Nature Reviews. Neuroscience |
| Rapaport AS, Schriewer J, Gilfillan S, et al. (2015) The Inhibitory Receptor NKG2A Sustains Virus-Specific CD8(+) T Cells in Response to a Lethal Poxvirus Infection. Immunity |