Volker Eulenburg

Affiliations: 
Universität Leipzig, Leipzig, Sachsen, Germany 
Area:
glycine, transporters, glycinergic synapses
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"Volker Eulenburg"
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Publications

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Groemer TW, Triller A, Zeilhofer HU, et al. (2022) Nociception in the Glycine Receptor Deficient Mutant Mouse Spastic. Frontiers in Molecular Neuroscience. 15: 832490
Eulenburg V, Hülsmann S. (2022) Synergistic Control of Transmitter Turnover at Glycinergic Synapses by GlyT1, GlyT2, and ASC-1. International Journal of Molecular Sciences. 23
Schulz D, Morschel J, Schuster S, et al. (2018) Inactivation of the Mouse L-Proline Transporter PROT Alters Glutamatergic Synapse Biochemistry and Perturbs Behaviors Required to Respond to Environmental Changes. Frontiers in Molecular Neuroscience. 11: 279
Kaczmarek-Hajek K, Zhang J, Kopp R, et al. (2018) Re-evaluation of neuronal P2X7 expression using novel mouse models and a P2X7-specific nanobody. Elife. 7
Eulenburg V, Knop G, Sedmak T, et al. (2018) GlyT1 determines the glycinergic phenotype of amacrine cells in the mouse retina. Brain Structure & Function
Kurolap A, Armbruster A, Hershkovitz T, et al. (2016) Loss of Glycine Transporter 1 Causes a Subtype of Glycine Encephalopathy with Arthrogryposis and Mildly Elevated Cerebrospinal Fluid Glycine. American Journal of Human Genetics
Zheng F, Puppel A, Huber SE, et al. (2015) Activin Controls Ethanol Potentiation of Inhibitory Synaptic Transmission through GABAA Receptors and Concomitant Behavioral Sedation. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology
Rahman J, Besser S, Schnell C, et al. (2015) Genetic ablation of VIAAT in glycinergic neurons causes a severe respiratory phenotype and perinatal death. Brain Structure & Function. 220: 2835-49
Lall D, Armbruster A, Ruffert K, et al. (2012) Transport activities and expression patterns of glycine transporters 1 and 2 in the developing murine brain stem and spinal cord. Biochemical and Biophysical Research Communications. 423: 661-6
Ruffert K, Himmel B, Lall D, et al. (2011) Glutamate residue 90 in the predicted transmembrane domain 2 is crucial for cation flux through channelrhodopsin 2. Biochemical and Biophysical Research Communications. 410: 737-43
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