Shanthini Sockanathan, Ph.D.

Affiliations: 
Neuroscience Johns Hopkins University School of Medicine, Baltimore, MD, United States 
Area:
nervous system development
Website:
http://neuroscience.jhu.edu/resources/directory/faculty/Shanthini-Sockanathan
Google:
"Shanthini Sockanathan"
Mean distance: 14.04 (cluster 11)
 
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Publications

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Westerhaus A, Joseph T, Meyers AJ, et al. (2022) The distribution and function of GDE2, a regulator of spinal motor neuron survival, are disrupted in Amyotrophic Lateral Sclerosis. Acta Neuropathologica Communications. 10: 73
Choi BR, Dobrowolski M, Sockanathan S. (2020) GDE2 expression in oligodendroglia regulates the pace of oligodendrocyte maturation. Developmental Dynamics : An Official Publication of the American Association of Anatomists
Choi BR, Cave C, Na CH, et al. (2020) GDE2-Dependent Activation of Canonical Wnt Signaling in Neurons Regulates Oligodendrocyte Maturation. Cell Reports. 31: 107540
Dobrowolski M, Cave C, Levy-Myers R, et al. (2020) GDE3 regulates oligodendrocyte precursor proliferation via release of soluble CNTFRα. Development (Cambridge, England)
Cave C, Sockanathan S. (2018) Transcription factor mechanisms guiding motor neuron differentiation and diversification. Current Opinion in Neurobiology. 53: 1-7
Cave C, Park S, Rodriguez M, et al. (2017) GDE2 is essential for neuronal survival in the postnatal mammalian spinal cord. Molecular Neurodegeneration. 12: 8
Cave C, Sockanathan S. (2016) Transcription Factor Hand-offs "Enhance" Motor Neuron Differentiation. Neuron. 92: 1149-1151
Na Y, Park S, Lee C, et al. (2016) Real-Time Imaging Reveals Properties of Glutamate-Induced Arc/Arg 3.1 Translation in Neuronal Dendrites. Neuron
Hatori Y, Yan Y, Schmidt K, et al. (2016) Neuronal differentiation is associated with a redox-regulated increase of copper flow to the secretory pathway. Nature Communications. 7: 10640
Yan Y, Wladyka C, Fujii J, et al. (2015) Prdx4 is a compartment-specific H2O2 sensor that regulates neurogenesis by controlling surface expression of GDE2. Nature Communications. 6: 7006
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