Susan Brain

Affiliations: 
King's College London, London, UK 
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"Susan Brain"
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Publications

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Zarban AA, Chaudhry H, de Sousa Valente J, et al. (2022) Elucidating the Ability of CGRP to Modulate Microvascular Events in Mouse Skin. International Journal of Molecular Sciences. 23
Thapa D, Barrett B, Argunhan F, et al. (2021) Influence of Cold-TRP Receptors on Cold-Influenced Behaviour. Pharmaceuticals (Basel, Switzerland). 15
Argunhan F, Thapa D, Aubdool AA, et al. (2021) Calcitonin Gene-Related Peptide Protects Against Cardiovascular Dysfunction Independently of Nitric Oxide In Vivo. Hypertension (Dallas, Tex. : 1979). HYPERTENSIONAHA12014
La Russa F, Lopes DM, Hobbs C, et al. (2019) Disruption of the Sensory System Affects Sterile Cutaneous Inflammation in vivo. The Journal of Investigative Dermatology
Kodji X, Arkless KL, Kee Z, et al. (2018) Sensory nerves mediate spontaneous behaviors in addition to inflammation in a murine model of psoriasis. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. fj201800395RR
Pereira DMS, Mendes SJF, Alawi K, et al. (2018) Transient Receptor Potential Canonical Channels 4 and 5 Mediate -Derived Thioredoxin Effects in Lipopolysaccharide-Injected Mice. Oxidative Medicine and Cellular Longevity. 2018: 4904696
Kemény Á, Kodji X, Horváth S, et al. (2018) TRPA1 acts in a protective manner in imiquimod-induced psoriasiform dermatitis in mice. The Journal of Investigative Dermatology
Aubdool AA, Argunhan F, Thakore P, et al. (2018) Response by Aubdool et al to Letters Regarding Article, "A Novel α-Calcitonin Gene-Related Peptide Analogue Protects Against End-Organ Damage in Experimental Hypertension, Cardiac Hypertrophy, and Heart Failure". Circulation. 137: 1201-1202
Wilde E, Aubdool AA, Thakore P, et al. (2017) Tail-Cuff Technique and Its Influence on Central Blood Pressure in the Mouse. Journal of the American Heart Association. 6
Aubdool AA, Thakore P, Argunhan F, et al. (2017) A Novel α-Calcitonin Gene-Related Peptide Analogue Protects Against End-Organ Damage in Experimental Hypertension, Cardiac Hypertrophy and Heart Failure. Circulation
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