Zachary A Sorrentino, PhD/MD
Affiliations: | University of Florida College of Medicine |
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| Quintin S, Sorrentino ZA, Mehkri Y, et al. (2022) Proteinopathies and Neurotrauma: Update on Degenerative Cascades. Jsm Neurosurgery and Spine. 9 |
| Sorrentino ZA, Hass E, Vijayaraghavan N, et al. (2022) Corrigendum to "Carboxy-terminal truncation and phosphorylation of α-synuclein elongates survival in a prion-like seeding mouse model of synucleinopathy" [Neurosci. Lett. 732 (2020) 1-11/135017]. Neuroscience Letters. 783: 136719 |
| Lloyd GM, Sorrentino ZA, Quintin S, et al. (2022) Unique seeding profiles and prion-like propagation of synucleinopathies are highly dependent on the host in human α-synuclein transgenic mice. Acta Neuropathologica |
| Hass EW, Sorrentino ZA, Xia Y, et al. (2021) Disease-, region- and cell type specific diversity of α-synuclein carboxy terminal truncations in synucleinopathies. Acta Neuropathologica Communications. 9: 146 |
| Dagra A, Miller DR, Lin M, et al. (2021) α-Synuclein-induced dysregulation of neuronal activity contributes to murine dopamine neuron vulnerability. Npj Parkinson's Disease. 7: 76 |
| Ferreira N, Gram H, Sorrentino ZA, et al. (2021) Multiple system atrophy-associated oligodendroglial protein p25α stimulates formation of novel α-synuclein strain with enhanced neurodegenerative potential. Acta Neuropathologica |
| Hass EW, Sorrentino ZA, Lloyd GM, et al. (2021) Robust α-synuclein pathology in select brainstem neuronal populations is a potential instigator of multiple system atrophy. Acta Neuropathologica Communications. 9: 80 |
| Williams T, Sorrentino Z, Weinrich M, et al. (2020) Differential cross-seeding properties of tau and α-synuclein in mouse models of tauopathy and synucleinopathy. Brain Communications. 2: fcaa090 |
| Trejo-Lopez JA, Sorrentino ZA, Riffe CJ, et al. (2020) Novel Monoclonal Antibodies Targeting the RRM2 domain of Human TDP-43 Protein. Neuroscience Letters. 135353 |
| Xia Y, Prokop S, Gorion KM, et al. (2020) Tau Ser208 phosphorylation promotes aggregation and reveals neuropathologic diversity in Alzheimer's disease and other tauopathies. Acta Neuropathologica Communications. 8: 88 |