Samuel E. Marsh
Affiliations: | University of California, Irvine, Irvine, CA |
Area:
Alzheimer's disease, stem cellsGoogle:
"https://scholar.google.com/citations?user=BxMhd9wAAAAJ&hl=en"Mean distance: 16.96 (cluster 32) | S | N | B | C | P |
Parents
Sign in to add mentor| Mathew M. Blurton-Jones | grad student | 2011-2016 | UC Irvine |
| Beth Stevens | post-doc | 2017- | Boston Children's Hospital/Harvard Medical School |
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Publications
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| Liddelow SA, Marsh SE, Stevens B. (2020) Microglia and Astrocytes in Disease: Dynamic Duo or Partners in Crime? Trends in Immunology |
| Hammond TR, Marsh SE, Stevens B. (2019) Immune Signaling in Neurodegeneration. Immunity. 50: 955-974 |
| Shukla AK, McIntyre LL, Marsh SE, et al. (2018) CD11a expression distinguishes infiltrating myeloid cells from plaque-associated microglia in Alzheimer's disease. Glia |
| Hammond TR, Dufort C, Dissing-Olesen L, et al. (2018) Single-Cell RNA Sequencing of Microglia throughout the Mouse Lifespan and in the Injured Brain Reveals Complex Cell-State Changes. Immunity |
| Mathur V, Burai R, Vest RT, et al. (2017) Activation of the STING-Dependent Type I Interferon Response Reduces Microglial Reactivity and Neuroinflammation. Neuron. 96: 1290-1302.e6 |
| Abud EM, Ramirez RN, Martinez ES, et al. (2017) iPSC-Derived Human Microglia-like Cells to Study Neurological Diseases. Neuron. 94: 278-293.e9 |
| Goldberg NR, Marsh SE, Ochaba J, et al. (2017) Human Neural Progenitor Transplantation Rescues Behavior and Reduces α-Synuclein in a Transgenic Model of Dementia with Lewy Bodies. Stem Cells Translational Medicine |
| Marsh SE, Blurton-Jones M. (2017) Neural stem cell therapy for neurodegenerative disorders: The role of neurotrophic support. Neurochemistry International |
| Marsh SE, Yeung ST, Torres M, et al. (2017) HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease. Stem Cell Reports. 8: 235-248 |
| Marsh SE, Abud EM, Lakatos A, et al. (2016) The adaptive immune system restrains Alzheimer's disease pathogenesis by modulating microglial function. Proceedings of the National Academy of Sciences of the United States of America |