Maj-Linda B. Selenica, Ph.D.
Affiliations: | 2019- | Sanders-Brown Center on Aging | University of Kentucky College of Medicine, Lexington, KY, United States |
Area:
AD, TDP-43, tau, brain metabolism, neuroinflammationWebsite:
http://selenicalab.createuky.netGoogle:
"Maj-Linda Selenica"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Marcia Gordon | post-doc | 2008-2013 | University of South Florida |
| Dave Morgan | post-doc | 2008-2013 | University of South Florida |
Children
Sign in to add trainee| Christopher Saunders | grad student | 2023- | University of Kentucky College of Medicine |
| Zainuddin Quadri | post-doc | 2015-2020 | University of Kentucky College of Medicine |
Collaborators
Sign in to add collaborator| Kevin Nash | collaborator | 2010- | University of Kentucky College of Medicine |
| Daniel C. Lee | collaborator | 2012- | University of Kentucky College of Medicine |
| Chad A. Dickey | collaborator | 2013- | |
| Donna M Wilcock | collaborator | 2019- | University of Kentucky |
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Loon A, Zamudio F, Sanneh A, et al. (2021) Accumulation of C-terminal cleaved tau is distinctly associated with cognitive deficits, synaptic plasticity impairment, and neurodegeneration in aged mice. Geroscience |
| Ma C, Hunt JB, Kovalenko A, et al. (2021) Myeloid Arginase 1 Insufficiency Exacerbates Amyloid-β Associated Neurodegenerative Pathways and Glial Signatures in a Mouse Model of Alzheimer's Disease: A Targeted Transcriptome Analysis. Frontiers in Immunology. 12: 628156 |
| Sandusky-Beltran LA, Kovalenko A, Placides DS, et al. (2021) Aberrant AZIN2 and polyamine metabolism precipitates tau neuropathology. The Journal of Clinical Investigation. 131 |
| Ma C, Hunt JB, Selenica MB, et al. (2020) Arginase 1 Insufficiency Precipitates Amyloid- Deposition and Hastens Behavioral Impairment in a Mouse Model of Amyloidosis. Frontiers in Immunology. 11: 582998 |
| Zamudio F, Loon AR, Smeltzer S, et al. (2020) TDP-43 mediated blood-brain barrier permeability and leukocyte infiltration promote neurodegeneration in a low-grade systemic inflammation mouse model. Journal of Neuroinflammation. 17: 283 |
| Smeltzer S, Quadri Z, Miller A, et al. (2020) Hypusination of Eif5a regulates cytoplasmic TDP-43 aggregation and accumulation in a stress-induced cellular model. Biochimica Et Biophysica Acta. Molecular Basis of Disease. 165939 |
| Joly-Amado A, Hunter J, Quadri Z, et al. (2020) CCL2 Overexpression in the Brain Promotes Glial Activation and Accelerates Tau Pathology in a Mouse Model of Tauopathy. Frontiers in Immunology. 11: 997 |
| Quadri Z, Johnson N, Zamudio F, et al. (2019) Overexpression of human wtTDP-43 causes impairment in hippocampal plasticity and behavioral deficits in CAMKII-tTa transgenic mouse model. Molecular and Cellular Neurosciences. 103418 |
| Sandusky-Beltran LA, Kovalenko A, Ma C, et al. (2019) Spermidine/spermine-N-acetyltransferase ablation impacts tauopathy-induced polyamine stress response. Alzheimer's Research & Therapy. 11: 58 |
| Grewal R, Haghighi M, Huang S, et al. (2016) Identifying biomarkers of dementia prevalent among amnestic mild cognitively impaired ethnic female patients. Alzheimer's Research & Therapy. 8: 43 |