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Hisashi Umemori

Affiliations: 
Harvard Medical School, Boston, MA, United States 
Area:
Synapse formation
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"Hisashi Umemori"
Mean distance: 14.74 (cluster 11)
 		SNBCP
Cross-listing: MichiganTree

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Parents

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Joshua R. Sanes post-doc Harvard

Children

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Siva Nagappan-Chettiar grad student Harvard Medical School
Aaron N. Reifler grad student University of Michigan

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Publications

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Barone I, Gilette NM, Hawks-Mayer H, et al. (2023) Synaptic BMAL1 phosphorylation controls circadian hippocampal plasticity. Science Advances. 9: eadj1010
Terauchi A, Yee P, Johnson-Venkatesh EM, et al. (2023) The projection-specific signals that establish functionally segregated dopaminergic synapses. Cell
Nagappan-Chettiar S, Burbridge TJ, Umemori H. (2023) Activity-Dependent Synapse Refinement: From Mechanisms to Molecules. The Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry. 10738584231170167
Nagappan-Chettiar S, Yasuda M, Johnson-Venkatesh EM, et al. (2023) The molecular signals that regulate activity-dependent synapse refinement in the brain. Current Opinion in Neurobiology. 79: 102692
Hoshina N, Johnson-Venkatesh EM, Hoshina M, et al. (2021) Female-specific synaptic dysfunction and cognitive impairment in a mouse model of disorder. Science (New York, N.Y.). 372
Yasuda M, Nagappan-Chettiar S, Johnson-Venkatesh EM, et al. (2021) An activity-dependent determinant of synapse elimination in the mammalian brain. Neuron
Li T, Chiou B, Gilman CK, et al. (2020) A splicing isoform of GPR56 mediates microglial synaptic refinement via phosphatidylserine binding. The Embo Journal. e104136
Luo L, Ambrozkiewicz MC, Benseler F, et al. (2020) Optimizing Nervous System-Specific Gene Targeting with Cre Driver Lines: Prevalence of Germline Recombination and Influencing Factors. Neuron
Terauchi A, Durlacher E, Pitino J, et al. (2019) Neuronal fibroblast growth factor 22 signaling during development, but not in adults, is involved in anhedonia. Neuroreport
Lehrman EK, Wilton DK, Litvina EY, et al. (2018) CD47 Protects Synapses from Excess Microglia-Mediated Pruning during Development. Neuron. 100: 120-134.e6
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