Tim Bartels
Affiliations: | 2009-2011 | Harvard Medical School, Boston, MA, United States |
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Parents
Sign in to add mentor| Klaus Beyer | grad student | 2005-2008 | Ludwig Maximilian University Munich |
| Dennis J. Selkoe | post-doc | 2009-2011 | Harvard Medical School |
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Publications
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| de Boni L, Wallis A, Hays Watson A, et al. (2024) Aggregation-resistant alpha-synuclein tetramers are reduced in the blood of Parkinson's patients. Embo Molecular Medicine |
| Fazzari M, Di Biase E, Zaccagnini L, et al. (2023) GM1 oligosaccharide efficacy against α-synuclein aggregation and toxicity in vitro. Biochimica Et Biophysica Acta. Molecular and Cell Biology of Lipids. 159350 |
| Ramalingam N, Jin SX, Moors TE, et al. (2023) Dynamic physiological α-synuclein S129 phosphorylation is driven by neuronal activity. Npj Parkinson's Disease. 9: 4 |
| de Boni L, Watson AH, Zaccagnini L, et al. (2022) Brain region-specific susceptibility of Lewy body pathology in synucleinopathies is governed by α-synuclein conformations. Acta Neuropathologica |
| Román-Vendrell C, Medeiros AT, Sanderson JB, et al. (2021) Effects of Excess Brain-Derived Human α-Synuclein on Synaptic Vesicle Trafficking. Frontiers in Neuroscience. 15: 639414 |
| Bartels T, De Schepper S, Hong S. (2020) Microglia modulate neurodegeneration in Alzheimer's and Parkinson's diseases. Science (New York, N.Y.). 370: 66-69 |
| Sanderson JB, De S, Jiang H, et al. (2020) Analysis of α-synuclein species enriched from cerebral cortex of humans with sporadic dementia with Lewy bodies. Brain Communications. 2: fcaa010 |
| Rovere M, Powers AE, Jiang H, et al. (2019) E46K-like α-synuclein mutants increase lipid interactions and disrupt membrane selectivity. The Journal of Biological Chemistry |
| Rovere M, Sanderson JB, Fonseca-Ornelas L, et al. (2018) Refolding of helical soluble α-synuclein through transient interaction with lipid interfaces. Febs Letters |
| Schapansky J, Khasnavis S, DeAndrade MP, et al. (2017) Familial knockin mutation of LRRK2 causes lysosomal dysfunction and accumulation of endogenous insoluble α-synuclein in neurons. Neurobiology of Disease |