John P. Norman

2007

Harris A. Gelbard	grad student	2007	Rochester
(The HIV-1 neurotoxin Tat rapidly induces the unfolded protein response and mitochondrial hyperpolarization: A unifying mechanism for neuropathogenesis and treatment.)

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Perry SW, Norman JP, Barbieri J, et al. (2011) Mitochondrial membrane potential probes and the proton gradient: a practical usage guide. Biotechniques. 50: 98-115
Norman JP, Perry SW, Reynolds HM, et al. (2008) HIV-1 Tat activates neuronal ryanodine receptors with rapid induction of the unfolded protein response and mitochondrial hyperpolarization. Plos One. 3: e3731
Norman JP, Perry SW, Kasischke KA, et al. (2007) HIV-1 trans activator of transcription protein elicits mitochondrial hyperpolarization and respiratory deficit, with dysregulation of complex IV and nicotinamide adenine dinucleotide homeostasis in cortical neurons. Journal of Immunology (Baltimore, Md. : 1950). 178: 869-76
Perry SW, Norman JP, Gelbard HA. (2005) Adjunctive therapies for HIV-1 associated neurologic disease. Neurotoxicity Research. 8: 161-6
Perry SW, Norman JP, Litzburg A, et al. (2005) HIV-1 transactivator of transcription protein induces mitochondrial hyperpolarization and synaptic stress leading to apoptosis. Journal of Immunology (Baltimore, Md. : 1950). 174: 4333-44
Perry SW, Norman JP, Litzburg A, et al. (2004) Antioxidants are required during the early critical period, but not later, for neuronal survival. Journal of Neuroscience Research. 78: 485-92