David Martinelli
Affiliations: | Stanford University, Palo Alto, CA |
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| Sticco MJ, Peña Palomino PA, Lukacsovich D, et al. (2021) C1QL3 promotes cell-cell adhesion by mediating complex formation between ADGRB3/BAI3 and neuronal pentraxins. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 35: e21194 |
| Chew KS, Fernandez DC, Hattar S, et al. (2017) Anatomical and Behavioral Investigation of C1ql3 in the Mouse Suprachiasmatic Nucleus. Journal of Biological Rhythms. 748730417704766 |
| Martinelli DC, Chew KS, Rohlmann A, et al. (2016) Expression of C1ql3 in Discrete Neuronal Populations Controls Efferent Synapse Numbers and Diverse Behaviors. Neuron. 91: 1034-51 |
| Ressl S, Vu BK, Vivona S, et al. (2015) Structures of C1q-like proteins reveal unique features among the C1q/TNF superfamily. Structure (London, England : 1993). 23: 688-99 |
| Aoto J, Martinelli DC, Malenka RC, et al. (2013) Presynaptic neurexin-3 alternative splicing trans-synaptically controls postsynaptic AMPA receptor trafficking. Cell. 154: 75-88 |
| Martinelli DC, Südhof TC. (2011) Cerebellins meet neurexins (Commentary on Matsuda & Yuzaki). The European Journal of Neuroscience. 33: 1445-6 |
| Bolliger MF, Martinelli DC, Südhof TC. (2011) The cell-adhesion G protein-coupled receptor BAI3 is a high-affinity receptor for C1q-like proteins. Proceedings of the National Academy of Sciences of the United States of America. 108: 2534-9 |