C. Blake Nichols
Affiliations: | 2010 | University of Washington, Seattle, Seattle, WA |
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"C. Nichols"Mean distance: 17.28 (cluster 32) | S | N | B | C | P |
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Sign in to add mentorStan McKnight | grad student | 2010 | University of Washington | |
(Sympathetic Stimulation of Adult Cardiomyocytes Requires Association of AKAP5 with a Sub-Population of L-Type Calcium Channels.) |
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Nichols CB, Chang CW, Ferrero M, et al. (2014) β-adrenergic signaling inhibits Gq-dependent protein kinase D activation by preventing protein kinase D translocation. Circulation Research. 114: 1398-409 |
Nystoriak MA, Nieves-Cintrón M, Nygren PJ, et al. (2014) AKAP150 contributes to enhanced vascular tone by facilitating large-conductance Ca2+-activated K+ channel remodeling in hyperglycemia and diabetes mellitus. Circulation Research. 114: 607-15 |
Jones BW, Brunet S, Gilbert ML, et al. (2012) Cardiomyocytes from AKAP7 knockout mice respond normally to adrenergic stimulation. Proceedings of the National Academy of Sciences of the United States of America. 109: 17099-104 |
Nichols CB, Rossow CF, Navedo MF, et al. (2010) Sympathetic stimulation of adult cardiomyocytes requires association of AKAP5 with a subpopulation of L-type calcium channels. Circulation Research. 107: 747-56 |
Weisenhaus M, Allen ML, Yang L, et al. (2010) Mutations in AKAP5 disrupt dendritic signaling complexes and lead to electrophysiological and behavioral phenotypes in mice. Plos One. 5: e10325 |
Nieves-Cintrón M, Amberg GC, Nichols CB, et al. (2007) Activation of NFATc3 down-regulates the beta1 subunit of large conductance, calcium-activated K+ channels in arterial smooth muscle and contributes to hypertension. The Journal of Biological Chemistry. 282: 3231-40 |