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monica di luca

Affiliations: 
University of Milan, Milano, Lombardia, Italy 
Area:
NMDA trafficking
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"monica di luca"
Mean distance: 17.86 (cluster 11)
 
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Daniela Mauceri grad student Università degli Studi di Milano

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Pavinato L, Stanic J, Barzasi M, et al. (2023) Missense variants in RPH3A cause defects in excitatory synaptic function and are associated with a clinically variable neurodevelopmental disorder. Genetics in Medicine : Official Journal of the American College of Medical Genetics. 100922
de la Fuente AG, Pelucchi S, Mertens J, et al. (2023) Novel therapeutic approaches to target neurodegeneration. British Journal of Pharmacology
Imarisio A, Ferrari E, Pilotto A, et al. (2022) Synaptic neurofilaments and GluN1-NfL interaction in experimental models of α-synucleinopathies. Neuro-Degenerative Diseases
Di Maio A, De Rosa A, Pelucchi S, et al. (2022) Analysis of mRNA and Protein Levels of , and Genes in Post-Mortem Brain of Schizophrenia, Parkinson's and Alzheimer's Disease Patients. International Journal of Molecular Sciences. 23
Pelucchi S, Gardoni F, Di Luca M, et al. (2022) Synaptic dysfunction in early phases of Alzheimer's Disease. Handbook of Clinical Neurology. 184: 417-438
Italia M, Ferrari E, Di Luca M, et al. (2021) GluA3-containing AMPA receptors: From physiology to synaptic dysfunction in brain disorders. Neurobiology of Disease. 161: 105539
Gardoni F, Di Luca M. (2021) Protein-protein interactions at the NMDA receptor complex: From synaptic retention to synaptonuclear protein messengers. Neuropharmacology. 108551
Gardoni F, Stanic J, Scheggia D, et al. (2021) NMDA and AMPA Receptor Autoantibodies in Brain Disorders: From Molecular Mechanisms to Clinical Features. Cells. 10
Pelucchi S, Vandermeulen L, Pizzamiglio L, et al. (2020) Cyclase-associated protein 2 dimerization regulates cofilin in synaptic plasticity and Alzheimer's disease. Brain Communications. 2: fcaa086
Bertan F, Wischhof L, Sosulina L, et al. (2020) Loss of Ryanodine Receptor 2 impairs neuronal activity-dependent remodeling of dendritic spines and triggers compensatory neuronal hyperexcitability. Cell Death and Differentiation
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