Year |
Citation |
Score |
2016 |
Carlson SL, Bohnsack JP, Morrow AL. Ethanol regulation of synaptic γ-aminobutyric acid (GABA)A α4 receptors is prevented by PKA activation. The Journal of Pharmacology and Experimental Therapeutics. PMID 26857960 DOI: 10.1124/Jpet.115.230417 |
0.699 |
|
2016 |
Bohnsack JP, Carlson SL, Morrow AL. Differential regulation of synaptic and extrasynaptic α4 GABA(A) receptor populations by protein kinase A and protein kinase C in cultured cortical neurons. Neuropharmacology. PMID 26767953 DOI: 10.1016/J.Neuropharm.2016.01.009 |
0.701 |
|
2015 |
Carlson SL, Bohnsack JP, Patel V, Morrow AL. Regulation of extrasynaptic GABAA α4 receptors by ethanol-induced PKA, but not PKC activation in cultured rat cerebral cortical neurons. The Journal of Pharmacology and Experimental Therapeutics. PMID 26483396 DOI: 10.1124/Jpet.115.228056 |
0.619 |
|
2014 |
Carlson SL, O'Buckley TK, Thomas R, Thiele TE, Morrow AL. Altered GABAA receptor expression and seizure threshold following acute ethanol challenge in mice lacking the RIIβ subunit of PKA. Neurochemical Research. 39: 1079-87. PMID 24104609 DOI: 10.1007/S11064-013-1167-0 |
0.553 |
|
2013 |
Fieber LA, Carlson SL, Kempsell AT, Greer JB, Schmale MC. Isolation of sensory neurons of Aplysia californica for patch clamp recordings of glutamatergic currents. Journal of Visualized Experiments : Jove. e50543. PMID 23892672 DOI: 10.3791/50543 |
0.609 |
|
2013 |
Carlson SL, Kumar S, Werner DF, Comerford CE, Morrow AL. Ethanol activation of protein kinase A regulates GABAA α1 receptor function and trafficking in cultured cerebral cortical neurons. The Journal of Pharmacology and Experimental Therapeutics. 345: 317-25. PMID 23408117 DOI: 10.1124/Jpet.112.201954 |
0.694 |
|
2012 |
Carlson SL, Kempsell AT, Fieber LA. Pharmacological evidence that D-aspartate activates a current distinct from ionotropic glutamate receptor currents in Aplysia californica. Brain and Behavior. 2: 391-401. PMID 22950043 DOI: 10.1002/Brb3.60 |
0.639 |
|
2012 |
Carlson SL, Fieber LA. Unique ionotropic receptors for D-aspartate are a target for serotonin-induced synaptic plasticity in Aplysia californica. Comparative Biochemistry and Physiology. Toxicology & Pharmacology : Cbp. 155: 151-9. PMID 21497673 DOI: 10.1016/J.Cbpc.2011.04.001 |
0.727 |
|
2011 |
Carlson SL, Fieber LA. Physiological evidence that D-aspartate activates a current distinct from ionotropic glutamate receptor currents in Aplysia californica neurons. Journal of Neurophysiology. 106: 1629-36. PMID 21753031 DOI: 10.1152/Jn.00403.2011 |
0.744 |
|
2010 |
Fieber LA, Carlson SL, Capo TR, Schmale MC. Changes in D-aspartate ion currents in the Aplysia nervous system with aging. Brain Research. 1343: 28-36. PMID 20452331 DOI: 10.1016/J.Brainres.2010.05.001 |
0.733 |
|
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