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Suman Chatterjee, Ph.D. - Publications

Affiliations: 
Biological Sciences Duquesne University, Pittsburgh, PA, United States 
Area:
Cell biology
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12 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2018 Yochum ZA, Cades J, Wang H, Chatterjee S, Simons BW, O'Brien JP, Khetarpal SK, Lemtiri-Chlieh G, Myers KV, Huang EH, Rudin CM, Tran PT, Burns TF. Targeting the EMT transcription factor TWIST1 overcomes resistance to EGFR inhibitors in EGFR-mutant non-small-cell lung cancer. Oncogene. PMID 30171258 DOI: 10.1038/S41388-018-0482-Y  0.34
2018 Han J, Goldstein LA, Hou W, Chatterjee S, Burns TF, Rabinowich H. HSP90 inhibition targets autophagy and induces a CASP9-dependent resistance mechanism in NSCLC. Autophagy. 1-14. PMID 29561705 DOI: 10.1080/15548627.2018.1434471  0.358
2018 Yochum ZA, Chatterjee S, Huang EH, Maurer DM, Attar MA, Dacic S, Stabile LP, Burns TF. Abstract 5889: TWIST1 is a key mediator of HGF-MET-driven resistance to targeted therapies in EGFR mutant and MET-driven lung cancer Cancer Research. 78: 5889-5889. DOI: 10.1158/1538-7445.Am2018-5889  0.364
2017 Chatterjee S, Burns TF. Targeting Heat Shock Proteins in Cancer: A Promising Therapeutic Approach. International Journal of Molecular Sciences. 18. PMID 28914774 DOI: 10.3390/Ijms18091978  0.307
2017 Yochum ZA, Cades J, Mazzacurati L, Neumann NM, Khetarpal SK, Chatterjee S, Wang H, Attar MA, Huang EH, Chatley SN, Nugent K, Somasundaram A, Engh J, Ewald AJ, Cho YJ, et al. A First-in-Class TWIST1 Inhibitor with Activity in Oncogene-driven Lung Cancer. Molecular Cancer Research : McR. PMID 28851812 DOI: 10.1158/1541-7786.Mcr-17-0298  0.368
2017 Chatterjee S, Huang EH, Christie I, Burns TF. Reactivation of the p90RSK-CDC25C pathway leads to bypass of the ganetespib induced G2/M arrest and mediates acquired resistance to ganetespib in KRAS mutant NSCLC. Molecular Cancer Therapeutics. PMID 28566436 DOI: 10.1158/1535-7163.Mct-17-0114  0.377
2017 Chatterjee S, Huang EH, Christie I, Kurland BF, Burns TF. Acquired resistance to the Hsp90 inhibitor, ganetespib in KRAS mutant NSCLC is mediated via reactivation of the ERK-p90RSK-mTOR signaling network. Molecular Cancer Therapeutics. PMID 28167505 DOI: 10.1158/1535-7163.Mct-16-0677  0.375
2017 Chatterjee S, Huang EH, Christie I, Burns TF. Abstract 4152: The p90RSK-CDC25C signaling pathway leads to bypass of the ganetespib induced G2/M arrest and mediates acquired resistance to ganetespib inKRASmutant NSCLC Cancer Research. 77: 4152-4152. DOI: 10.1158/1538-7445.Am2017-4152  0.322
2016 Chatterjee S, Huang EH, Burns TF. Abstract 2942: p90 Ribosomal S6 kinase mediates acquired resistance to ganetespib in KRAS mutant NSCLC Cancer Research. 76: 2942-2942. DOI: 10.1158/1538-7445.Am2016-2942  0.388
2015 Chatterjee S, Burns TF. Abstract 762: Ganetespib resistance inKRASmutant NSCLC is mediated through bypassing the G2/M checkpoint and reactivating the PI3K/MTOR pathway Cancer Research. 75: 762-762. DOI: 10.1158/1538-7445.Am2015-762  0.398
2014 Chatterjee S, Elinson RP. Commitment to nutritional endoderm in Eleutherodactylus coqui involves altered nodal signaling and global transcriptional repression. Journal of Experimental Zoology. Part B, Molecular and Developmental Evolution. 322: 27-44. PMID 24323742 DOI: 10.1002/Jez.B.22543  0.508
2011 Chatterjee S, Elinson R. Differential EcSmad2 expression in early development of the direct developing frog Eleutherodactylus coqui Developmental Biology. 356: 247. DOI: 10.1016/J.Ydbio.2011.05.548  0.485
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