Year |
Citation |
Score |
2020 |
Patel MY, Stovall K, Franklin JL. The intrinsic apoptotic pathway lies upstream of oxidative stress in multiple organs. Free Radical Biology & Medicine. PMID 32659346 DOI: 10.1016/j.freeradbiomed.2020.05.025 |
0.31 |
|
2016 |
McManus MJ, Franklin JL. Dissociation of JNK Activation from Elevated Levels of Reactive Oxygen Species, Cytochrome c Release, and Cell Death in NGF-Deprived Sympathetic Neurons. Molecular Neurobiology. PMID 27957682 DOI: 10.1007/s12035-016-0332-2 |
0.425 |
|
2015 |
Kirkland RA, Franklin JL. Bax and caspases regulate increased production of mitochondria-derived reactive species in neuronal apoptosis: LACK of A role for depletion of cytochrome c from the mitochondrial electron transport chain. Biochemistry and Biophysics Reports. 4: 158-168. PMID 29124200 DOI: 10.1016/j.bbrep.2015.09.004 |
0.383 |
|
2014 |
McManus MJ, Murphy MP, Franklin JL. Mitochondria-derived reactive oxygen species mediate caspase-dependent and -independent neuronal deaths. Molecular and Cellular Neurosciences. 63: 13-23. PMID 25239010 DOI: 10.1016/j.mcn.2014.09.002 |
0.437 |
|
2011 |
McManus MJ, Murphy MP, Franklin JL. The mitochondria-targeted antioxidant MitoQ prevents loss of spatial memory retention and early neuropathology in a transgenic mouse model of Alzheimer's disease. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 15703-15. PMID 22049413 DOI: 10.1523/JNEUROSCI.0552-11.2011 |
0.331 |
|
2011 |
Franklin JL. Redox regulation of the intrinsic pathway in neuronal apoptosis. Antioxidants & Redox Signaling. 14: 1437-48. PMID 20812874 DOI: 10.1089/ars.2010.3596 |
0.41 |
|
2010 |
Kirkland RA, Saavedra GM, Cummings BS, Franklin JL. Bax regulates production of superoxide in both apoptotic and nonapoptotic neurons: role of caspases. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 16114-27. PMID 21123558 DOI: 10.1523/Jneurosci.2862-10.2010 |
0.382 |
|
2007 |
Kirkland RA, Saavedra GM, Franklin JL. Rapid activation of antioxidant defenses by nerve growth factor suppresses reactive oxygen species during neuronal apoptosis: evidence for a role in cytochrome c redistribution. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 27: 11315-26. PMID 17942726 DOI: 10.1523/JNEUROSCI.3590-07.2007 |
0.4 |
|
2007 |
Kirkland RA, Franklin JL. Bax affects production of reactive oxygen by the mitochondria of non-apoptotic neurons. Experimental Neurology. 204: 458-61. PMID 17097638 DOI: 10.1016/j.expneurol.2006.09.013 |
0.443 |
|
2003 |
Kirkland RA, Franklin JL. Prooxidant effects of NGF withdrawal and MEK inhibition in sympathetic neurons. Antioxidants & Redox Signaling. 5: 635-9. PMID 14580320 DOI: 10.1089/152308603770310301 |
0.388 |
|
2003 |
Kirkland RA, Franklin JL. Bax, reactive oxygen, and cytochrome c release in neuronal apoptosis. Antioxidants & Redox Signaling. 5: 589-96. PMID 14580315 DOI: 10.1089/152308603770310257 |
0.43 |
|
2003 |
Franklin JL. Programmed neuronal death. Antioxidants & Redox Signaling. 5: 583-7. PMID 14580314 DOI: 10.1089/152308603770310248 |
0.319 |
|
2002 |
Kirkland RA, Adibhatla RM, Hatcher JF, Franklin JL. Loss of cardiolipin and mitochondria during programmed neuronal death: evidence of a role for lipid peroxidation and autophagy. Neuroscience. 115: 587-602. PMID 12421624 DOI: 10.1016/S0306-4522(02)00512-2 |
0.341 |
|
2002 |
Kirkland RA, Windelborn JA, Kasprzak JM, Franklin JL. A Bax-induced pro-oxidant state is critical for cytochrome c release during programmed neuronal death. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 22: 6480-90. PMID 12151527 DOI: 10.1523/Jneurosci.22-15-06480.2002 |
0.373 |
|
2001 |
Kirkland RA, Franklin JL. Evidence for redox regulation of cytochrome C release during programmed neuronal death: antioxidant effects of protein synthesis and caspase inhibition. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 21: 1949-63. PMID 11245680 |
0.324 |
|
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